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International Journal of Molecular Sciences|February 3, 2021
USP2-Related Cellular Signaling and Consequent Pathophysiological OutcomesHiroshi Kitamura, Mayuko HashimotoBiochemical and Biophysical Research Communications|May 30, 2019
High temperatures promote cell-to-cell plasmid transformation in Escherichia coliMayuko Hashimoto, Haruka Hasegawa, Sumio MaedaCurrent Research in Microbial Sciences|August 1, 2022
Subminimal inhibitory concentrations of ampicillin and mechanical stimuli cooperatively promote cell-to-cell plasmid transformation in Escherichia coliSayuri Kasagaki, Mayuko Hashimoto, Sumio MaedaClinical, Cosmetic and Investigational Dermatology|December 26, 2024
Combination Treatment with Cryolipolysis and Hyaluronic Acid Fillers for Jawline EnhancementKumiko Shimojima, Mayuko Hashimoto, Yuhi Yamaoka, et al.Chemistry, an Asian Journal|September 9, 2014
Mechanochromic luminescence characteristics of pyridine-terminated chromophores in the solid state and in a poly(vinyl alcohol) matrixMizuho Kondo, Seiya Miura, Kentaro Okumoto, et al.International Journal of Molecular Sciences|November 27, 2024
USP2 Mitigates Reactive Oxygen Species-Induced Mitochondrial Damage via UCP2 Expression in MyoblastsHiroshi Kitamura, Masaki Fujimoto, Mayuko Hashimoto, et al.Chemistry, an Asian Journal|January 3, 2019
Controlling the Emergence and Shift Direction of Mechanochromic Luminescence Color of a Pyridine-Terminated CompoundMizuho Kondo, Taku Yamoto, Seiya Miura, et al.Biomedical Research (Tokyo, Japan)|April 20, 2018
Establishment of protocol for preparation of gene-edited bovine ear-derived fibroblasts for somatic cell nuclear transplantationTakeshi Ishino, Mayuko Hashimoto, Misato Amagasa, et al.Experimental Animals|July 21, 2025
Suppression of USP2 in mouse skeletal muscle: a model of oxidative stress in muscle tissueMasaki Fujimoto, Tomohito Iwasaki, Marina Hosotani Saito, et al.Physiological Reports|July 30, 2019
Inhibition of ubiquitin-specific protease 2 causes accumulation of reactive oxygen species, mitochondria dysfunction, and intracellular ATP decrement in C2C12 myoblastsMayuko Hashimoto, Natsuko Saito, Haru Ohta, et al.Pageof 2