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Melanie Grosch

Showing results (1-10 of 8) with videos related to

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EXCLI Journal|June 15, 2016
Selenoprotein M is expressed during bone developmentMelanie Grosch, Jennifer Fuchs, Michael Bösl, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|August 16, 2011
Ucmaa (Grp-2) is required for zebrafish skeletal development. Evidence for a functional role of its glutamate γ-carboxylationCristian Dan Neacsu, Melanie Grosch, Mercedes Tejada, et al.
Journal of Virology|February 28, 2006
Global suppression of the host antiviral response by Ebola- and Marburgviruses: increased antagonism of the type I interferon response is associated with enhanced virulenceJohn C Kash, Elke Mühlberger, Victoria Carter, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|May 14, 2013
Identification of a Ninein (NIN) mutation in a family with spondyloepimetaphyseal dysplasia with joint laxity (leptodactylic type)-like phenotypeMelanie Grosch, Barbara Grüner, Stephanie Spranger, et al.
Kidney International Reports|January 16, 2026
<i>ACTN4</i> p.Ile150Met Causes FSGS With Validation in Primary Fibroblasts and Immortalized PodocytesMelanie Grosch, Jan René Haak, Cathiana Kolb, et al.
American Journal of Physiology. Renal Physiology|March 15, 2018
Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8Katy Billot, Charlène Coquil, Benoit Villiers, et al.
Journal of Cell Science|August 4, 2021
A polycystin-2 protein with modified channel properties leads to an increased diameter of renal tubules and to renal cystsMelanie Grosch, Katrin Brunner, Alexandr V Ilyaskin, et al.
Kidney International Reports|March 9, 2026
<i>MYH9</i> Variant p.(Arg424Gly) Alters Nonmuscle Myosin IIA Contraction, Causing Atypical <i>MYH9</i>-related DiseaseLena Pollinger, Johannes N Greve, Melanie Grosch, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
EXCLI Journal|June 15, 2016
Selenoprotein M is expressed during bone developmentMelanie Grosch, Jennifer Fuchs, Michael Bösl, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|August 16, 2011
Ucmaa (Grp-2) is required for zebrafish skeletal development. Evidence for a functional role of its glutamate γ-carboxylationCristian Dan Neacsu, Melanie Grosch, Mercedes Tejada, et al.
Journal of Virology|February 28, 2006
Global suppression of the host antiviral response by Ebola- and Marburgviruses: increased antagonism of the type I interferon response is associated with enhanced virulenceJohn C Kash, Elke Mühlberger, Victoria Carter, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|May 14, 2013
Identification of a Ninein (NIN) mutation in a family with spondyloepimetaphyseal dysplasia with joint laxity (leptodactylic type)-like phenotypeMelanie Grosch, Barbara Grüner, Stephanie Spranger, et al.
Kidney International Reports|January 16, 2026
<i>ACTN4</i> p.Ile150Met Causes FSGS With Validation in Primary Fibroblasts and Immortalized PodocytesMelanie Grosch, Jan René Haak, Cathiana Kolb, et al.
American Journal of Physiology. Renal Physiology|March 15, 2018
Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8Katy Billot, Charlène Coquil, Benoit Villiers, et al.
Journal of Cell Science|August 4, 2021
A polycystin-2 protein with modified channel properties leads to an increased diameter of renal tubules and to renal cystsMelanie Grosch, Katrin Brunner, Alexandr V Ilyaskin, et al.
Kidney International Reports|March 9, 2026
<i>MYH9</i> Variant p.(Arg424Gly) Alters Nonmuscle Myosin IIA Contraction, Causing Atypical <i>MYH9</i>-related DiseaseLena Pollinger, Johannes N Greve, Melanie Grosch, et al.
Pageof 1