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Plos Genetics|May 10, 2011
Reciprocal sign epistasis between frequently experimentally evolved adaptive mutations causes a rugged fitness landscapeDaniel J Kvitek, Gavin SherlockEukaryotic Cell|August 6, 2013
Ras signaling gets fine-tuned: regulation of multiple pathogenic traits of Candida albicansDiane O Inglis, Gavin SherlockPlos Biology|February 4, 2017
Seeking Goldilocks During Evolution of Drug ResistanceGavin Sherlock, Dmitri A PetrovTrends in Biotechnology|May 26, 2004
Final words: cell age and cell cycle are unlinkedPaul T Spellman, Gavin SherlockYeast (Chichester, England)|January 24, 2025
The Viral K1 Killer Yeast System: Toxicity, Immunity, and ResistanceAngelina Chan, Michelle Hays, Gavin SherlockJournal of Molecular Evolution|March 6, 2023
Fit-Seq2.0: An Improved Software for High-Throughput Fitness Measurements Using Pooled Competition AssaysFangfei Li, Jason Tarkington, Gavin SherlockBMC Genomics|May 6, 2023
An improved algorithm for inferring mutational parameters from bar-seq evolution experimentsFangfei Li, Aditya Mahadevan, Gavin SherlockPlos Genetics|January 25, 2021
Adaptation is influenced by the complexity of environmental change during evolution in a dynamic environmentSébastien Boyer, Lucas Hérissant, Gavin SherlockTrends in Cell Biology|May 15, 2023
Experimental evolution for cell biologyJana Helsen, Gavin Sherlock, Gautam DeyNature Communications|September 1, 2021
Changes in the distribution of fitness effects and adaptive mutational spectra following a single first step towards adaptationDimitra Aggeli, Yuping Li, Gavin SherlockPageof 17