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DNA Repair|October 24, 2018
Inactive Atm abrogates DSB repair in mouse cerebellum more than does Atm loss, without causing a neurological phenotypeEfrat Tal, Marina Alfo, Shan Zha, et al.Ageing Research Reviews|June 1, 2022
The hallmarks of aging in Ataxia-TelangiectasiaJulio Aguado, Cecilia Gómez-Inclán, Hannah C Leeson, et al.Cells|October 27, 2022
Genomic Amplification of UBQLN4 Is a Prognostic and Treatment Resistance FactorYuta Kobayashi, Matias A Bustos, Yoshiaki Shoji, et al.BMC Bioinformatics|September 24, 2005
EXPANDER--an integrative program suite for microarray data analysisRon Shamir, Adi Maron-Katz, Amos Tanay, et al.The American Journal of Pathology|July 19, 2011
A role for vascular deficiency in retinal pathology in a mouse model of ataxia-telangiectasiaDorit Raz-Prag, Ronit Galron, Niva Segev-Amzaleg, et al.Methods in Molecular Biology (Clifton, N.J.)|November 9, 2017
Upgrading Affinity Screening Experiments by Analysis of Next-Generation Sequencing DataChristian Grohmann, Michael BlankOncogene|February 19, 2002
ATM-dependent activation of the gene encoding MAP kinase phosphatase 5 by radiomimetic DNA damageAnat Bar-Shira, Sharon Rashi-Elkeles, Liat Zlochover, et al.Molecular Cell|July 29, 2008
ATM signaling facilitates repair of DNA double-strand breaks associated with heterochromatinAaron A Goodarzi, Angela T Noon, Dorothee Deckbar, et al.Current Opinion in Chemical Biology|July 12, 2005
Aptamers as tools for target validationMichael Blank, Michael BlindCancer Letters|November 7, 2016
Targeting p38 MAP kinase signaling in cancer through post-translational modificationsXiao Zou, Michael BlankPageof 17