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American Journal of Medical Genetics. Part A|July 17, 2007
Ataxia-telangiectasia: mild neurological presentation despite null ATM mutation and severe cellular phenotypeNeora Alterman, Aviva Fattal-Valevski, Lilach Moyal, et al.Molecular Therapy. Nucleic Acids|January 24, 2017
Aptamer Selection Technology and Recent AdvancesMichael Blind, Michael BlankAging Cell|May 31, 2023
Accelerated replicative senescence of ataxia-telangiectasia skin fibroblasts is retained at physiologic oxygen levels, with unique and common transcriptional patternsMajd Haj, Amit Levon, Yann Frey, et al.Science Signaling|December 9, 2010
ATM-dependent and -independent dynamics of the nuclear phosphoproteome after DNA damageAriel Bensimon, Alexander Schmidt, Yael Ziv, et al.Nucleic Acids Research|April 10, 2015
The COP9 signalosome is vital for timely repair of DNA double-strand breaksMichal Meir, Yaron Galanty, Lior Kashani, et al.Molecular Biology of the Cell|July 4, 2008
WRN is required for ATM activation and the S-phase checkpoint in response to interstrand cross-link-induced DNA double-strand breaksWen-Hsing Cheng, Diana Muftic, Meltem Muftuoglu, et al.Analytical Biochemistry|May 13, 2015
Functional analysis of protein ubiquitinationGal Levy-Cohen, Michael BlankGenome Biology|May 17, 2005
Dissection of a DNA-damage-induced transcriptional network using a combination of microarrays, RNA interference and computational promoter analysisRan Elkon, Sharon Rashi-Elkeles, Yaniv Lerenthal, et al.The EMBO Journal|October 24, 2009
Optimal function of the DNA repair enzyme TDP1 requires its phosphorylation by ATM and/or DNA-PKBenu Brata Das, Smitha Antony, Shalu Gupta, et al.Community Mental Health Journal|June 16, 2010
Case managers discovering what recovery means through an HIV prevention interventionJulie Tennille, Phyllis Solomon, Michael BlankPageof 17