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Journal of Virology|August 2, 2022
Regulation of HPV E7 Stability by E6-Associated Protein (E6AP)Arushi Vats, Oscar Trejo-Cerro, Paola Massimi, et al.Oncogene|July 27, 2005
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2ADavid Pim, Paola Massimi, Stephen M Dilworth, et al.Virology|December 26, 2014
High risk HPV E6 oncoproteins impair the subcellular distribution of the four and a half LIM-only protein 2 (FHL2)Joaquin Manzo-Merino, Paola Massimi, Lawrence Banks, et al.Plos One|January 30, 2013
A novel interaction between hScrib and PP1γ downregulates ERK signaling and suppresses oncogene-induced cell transformationKazunori Nagasaka, Takayuki Seiki, Aki Yamashita, et al.Journal of Cellular and Molecular Medicine|December 22, 2018
Oncogenic comparison of human papillomavirus type 58 E7 variantsPriscilla Ty Law, Siaw Shi Boon, Chenghua Hu, et al.Plos Pathogens|May 23, 2019
The HPV-18 E7 CKII phospho acceptor site is required for maintaining the transformed phenotype of cervical tumour-derived cellsOm Basukala, Suruchi Mittal, Paola Massimi, et al.Journal of Cellular Biochemistry|April 23, 2002
Human papillomavirus-16 E7 protein inhibits the DNA interaction of the TATA binding transcription factorEdio Maldonado, María Eugenia Cabrejos, Lawrence Banks, et al.Tumour Virus Research|March 14, 2024
HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complexOscar Trejo-Cerro, Om Basukala, Michael P Myers, et al.Scientific Reports|January 18, 2018
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entryJustyna Broniarczyk, Nadja Ring, Paola Massimi, et al.Journal of Virology|May 27, 2026
Rethinking residual p53 function in HPV-positive cervical cancer cellsSannia Farrukh, Nezka Kavcic, Giulia Canarutto, et al.Pageof 18