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Molecular Cell|June 12, 2012
RNF20 and USP44 regulate stem cell differentiation by modulating H2B monoubiquitylationGilad Fuchs, Efrat Shema, Rita Vesterman, et al.
The EMBO Journal|June 10, 2005
TAp63alpha induces apoptosis by activating signaling via death receptors and mitochondriaOlav Gressner, Tobias Schilling, Katja Lorenz, et al.
Molecular Cell|May 21, 2011
Two phases of mitogenic signaling unveil roles for p53 and EGR1 in elimination of inconsistent growth signalsYaara Zwang, Aldema Sas-Chen, Yotam Drier, et al.
FEBS Letters|November 29, 2022
miR-4734 conditionally suppresses ER stress-associated proinflammatory responsesDan Michael, Ester Feldmesser, Chagay Gonen, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 28, 2003
The p53-inducible TSAP6 gene product regulates apoptosis and the cell cycle and interacts with Nix and the Myt1 kinaseBrent J Passer, Vanessa Nancy-Portebois, Nathalie Amzallag, et al.
Nature Communications|November 28, 2022
Breast cancer plasticity is restricted by a LATS1-NCOR1 repressive axisYael Aylon, Noa Furth, Giuseppe Mallel, et al.
Gastroenterology|April 12, 2016
Genomic Alterations Observed in Colitis-Associated Cancers Are Distinct From Those Found in Sporadic Colorectal Cancers and Vary by Type of Inflammatory Bowel DiseaseRona Yaeger, Manish A Shah, Vincent A Miller, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 16, 2014
Pla2g16 phospholipase mediates gain-of-function activities of mutant p53Shunbin Xiong, Huolin Tu, Madhusudhan Kollareddy, et al.
Blood|April 15, 2018
TRRAP is essential for regulating the accumulation of mutant and wild-type p53 in lymphomaAlexander Jethwa, Mikołaj Słabicki, Jennifer Hüllein, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 5, 2021
TP53 missense mutations in PDAC are associated with enhanced fibrosis and an immunosuppressive microenvironmentMartino Maddalena, Giuseppe Mallel, Nishanth Belugali Nataraj, et al.
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