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Experimental Neurology|June 2, 2000
Apolipoprotein E epsilon4 allele has no effect on age at onset or duration of disease in cases of frontotemporal dementia with pick- or microvacuolar-type histologyS M Pickering-Brown, F Owen, A Isaacs, et al.
The American Journal of Pathology|April 1, 1996
Predominant deposition of amyloid-beta 42(43) in plaques in cases of Alzheimer's disease and hereditary cerebral hemorrhage associated with mutations in the amyloid precursor protein geneD M Mann, T Iwatsubo, Y Ihara, et al.
Insect Biochemistry and Molecular Biology|March 27, 2001
[3H]-Methyllycaconitine: a high affinity radioligand that labels invertebrate nicotinic acetylcholine receptorsR J Lind, D J Hardick, I S Blagbrough, et al.
Brain : a Journal of Neurology|September 1, 1999
The impact of different presenilin 1 andpresenilin 2 mutations on amyloid deposition, neurofibrillary changes and neuronal loss in the familial Alzheimer's disease brain: evidence for other phenotype-modifying factorsT Gómez-Isla, W B Growdon, M J McNamara, et al.
American Journal of Medical Genetics|January 23, 2002
Alzheimer disease is not associated with polymorphisms in the angiotensinogen and renin genesA Taylor, M Ezquerra, G Bagri, et al.
The Journal of General Virology|July 1, 1996
Enterovirus infection of the central nervous system of humans: lack of association with chronic neurological diseaseP Muir, F Nicholson, G T Spencer, et al.
Annals of Neurology|August 1, 1996
Amyloid beta protein (Abeta) deposition in chromosome 14-linked Alzheimer's disease: predominance of Abeta42(43)D M Mann, T Iwatsubo, N J Cairns, et al.
Neuropathology and Applied Neurobiology|April 14, 2012
Nuclear carrier and RNA-binding proteins in frontotemporal lobar degeneration associated with fused in sarcoma (FUS) pathological changesY S Davidson, A C Robinson, Q Hu, et al.
Neurology|January 3, 2001
Multiple system atrophy/progressive supranuclear palsy: alpha-Synuclein, synphilin, tau, and APOEH R Morris, J R Vaughan, S R Datta, et al.
The American Journal of Pathology|November 12, 1998
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genesC F Lippa, H Fujiwara, D M Mann, et al.
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