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European Journal of Histochemistry : EJH|June 3, 2026
Elevated caspase 3 expression correlates with severe inflammation in Crohn's diseaseNafie Sedju, Michael Naumann, Michael ViethGut|March 24, 2012
Protein kinase C isozymes regulate matrix metalloproteinase-1 expression and cell invasion in Helicobacter pylori infectionOlga Sokolova, Michael Vieth, Michael NaumannHistochemistry and Cell Biology|December 31, 2024
Differential expression of the ubiquitin-editing enzyme A20 in gastric biopsies indicates the severity of diseaseStephan Schnizler, Michael Naumann, Michael ViethTrends in Molecular Medicine|January 11, 2022
Helicobacter pylori-induced NF-κB: trailblazer for gastric pathophysiologyGunter Maubach, Michael Vieth, Francesco Boccellato, et al.The International Journal of Biochemistry & Cell Biology|September 8, 2014
Helicobacter pylori promotes eukaryotic protein translation by activating phosphatidylinositol 3 kinase/mTOROlga Sokolova, Michael Vieth, Thorsten Gnad, et al.Biochimica Et Biophysica Acta. Molecular Cell Research|January 14, 2018
Helicobacter pylori induces direct activation of the lymphotoxin beta receptor and non-canonical nuclear factor-kappa B signalingMichael Hartmut Feige, Michael Vieth, Olga Sokolova, et al.The Journal of Biological Chemistry|February 5, 2005
Inhibition of RICK/nuclear factor-kappaB and p38 signaling attenuates the inflammatory response in a murine model of Crohn diseaseEike Hollenbach, Michael Vieth, Albert Roessner, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 4, 2004
Inhibition of p38 MAP kinase- and RICK/NF-kappaB-signaling suppresses inflammatory bowel diseaseEike Hollenbach, Manfred Neumann, Michael Vieth, et al.Best Practice & Research. Clinical Gastroenterology|July 29, 2008
Contribution of histology to the diagnosis of reflux diseaseMichael ViethDigestion|October 10, 2008
Structural abnormalities of endoscopy-negative reflux disease--real or perceived?Michael ViethPageof 65