Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Nancy I Kerkvliet

Showing results (1-10 of 44) with videos related to

Pageof 5
Sort By:
Toxicologic Pathology|November 18, 2011
TCDD: an environmental immunotoxicant reveals a novel pathway of immunoregulation--a 30-year odysseyNancy I Kerkvliet
Biochemical Pharmacology|December 23, 2008
AHR-mediated immunomodulation: the role of altered gene transcriptionNancy I Kerkvliet
International Immunopharmacology|January 29, 2002
Recent advances in understanding the mechanisms of TCDD immunotoxicityNancy I Kerkvliet
Current Opinion in Toxicology|September 26, 2017
Is chronic AhR activation by rapidly metabolized ligands safe for the treatment of immune-mediated diseases?Allison K Ehrlich, Nancy I Kerkvliet
Annals of the New York Academy of Sciences|February 12, 2010
Dioxin and immune regulation: emerging role of aryl hydrocarbon receptor in the generation of regulatory T cellsNikki B Marshall, Nancy I Kerkvliet
Current Protocols in Toxicology|October 10, 2012
Immune cell phenotyping using flow cytometryJulie A Oughton, Nancy I Kerkvliet
Molecular Pharmacology|August 16, 2002
2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses tumor necrosis factor-alpha and anti-CD40-induced activation of NF-kappaB/Rel in dendritic cells: p50 homodimer activation is not affectedCarl E Ruby, Mark Leid, Nancy I Kerkvliet
International Immunopharmacology|April 12, 2003
Functional alterations in CD11b(+)Gr-1(+) cells in mice injected with allogeneic tumor cells and treated with 2,3,7,8-tetrachlorodibenzo-p-dioxinJin-Young Choi, Julie A Oughton, Nancy I Kerkvliet
Journal of Immunotoxicology|April 3, 2008
2,3,7,8-Tetrachlorodibenzo-p-dioxin alters the differentiation of alloreactive CD8+ T cells toward a regulatory T cell phenotype by a mechanism that is dependent on aryl hydrocarbon receptor in CD4+ T cellsCastle J Funatake, Nikki B Marshall, Nancy I Kerkvliet
Toxicology and Applied Pharmacology|December 20, 2002
T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDDNancy I Kerkvliet, David M Shepherd, Linda Baecher-Steppan
Pageof 5

Showing results (1-10 of 44) with videos related to

Sort By:
Pageof 5
Toxicologic Pathology|November 18, 2011
TCDD: an environmental immunotoxicant reveals a novel pathway of immunoregulation--a 30-year odysseyNancy I Kerkvliet
Biochemical Pharmacology|December 23, 2008
AHR-mediated immunomodulation: the role of altered gene transcriptionNancy I Kerkvliet
International Immunopharmacology|January 29, 2002
Recent advances in understanding the mechanisms of TCDD immunotoxicityNancy I Kerkvliet
Current Opinion in Toxicology|September 26, 2017
Is chronic AhR activation by rapidly metabolized ligands safe for the treatment of immune-mediated diseases?Allison K Ehrlich, Nancy I Kerkvliet
Annals of the New York Academy of Sciences|February 12, 2010
Dioxin and immune regulation: emerging role of aryl hydrocarbon receptor in the generation of regulatory T cellsNikki B Marshall, Nancy I Kerkvliet
Current Protocols in Toxicology|October 10, 2012
Immune cell phenotyping using flow cytometryJulie A Oughton, Nancy I Kerkvliet
Molecular Pharmacology|August 16, 2002
2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses tumor necrosis factor-alpha and anti-CD40-induced activation of NF-kappaB/Rel in dendritic cells: p50 homodimer activation is not affectedCarl E Ruby, Mark Leid, Nancy I Kerkvliet
International Immunopharmacology|April 12, 2003
Functional alterations in CD11b(+)Gr-1(+) cells in mice injected with allogeneic tumor cells and treated with 2,3,7,8-tetrachlorodibenzo-p-dioxinJin-Young Choi, Julie A Oughton, Nancy I Kerkvliet
Journal of Immunotoxicology|April 3, 2008
2,3,7,8-Tetrachlorodibenzo-p-dioxin alters the differentiation of alloreactive CD8+ T cells toward a regulatory T cell phenotype by a mechanism that is dependent on aryl hydrocarbon receptor in CD4+ T cellsCastle J Funatake, Nikki B Marshall, Nancy I Kerkvliet
Toxicology and Applied Pharmacology|December 20, 2002
T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDDNancy I Kerkvliet, David M Shepherd, Linda Baecher-Steppan
Pageof 5