Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Nathan Campbell

Showing results (11-20 of 40) with videos related to

Pageof 4
Sort By:
Hypertension in Pregnancy|May 3, 2026
Placental CD4⁺ T cells from women with gestational diabetes recapitulate disease features in a pregnant rat model, improved by metformin or mitotempoDanielle Frieson, Jean Vel, Tyler Johnson, et al.
Cells|November 27, 2021
Progesterone Induced Blocking Factor Reduces Hypertension and Placental Mitochondrial Dysfunction in Response to sFlt-1 during PregnancyEvangeline Deer, Jalisa Jones, Denise C Cornelius, et al.
Pregnancy Hypertension|March 7, 2021
The role of tumor necrosis factor in triggering activation of natural killer cell, multi-organ mitochondrial dysfunction and hypertension during pregnancyAswathi Jayaram, Evangeline Deer, Lorena M Amaral, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|October 28, 2016
Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancyDenise C Cornelius, Lorena M Amaral, Kedra Wallace, et al.
International Journal of Cerebrovascular Disease and Stroke|October 30, 2023
AT1-AA Infusion during Pregnancy Impairs CBF Autoregulation PostpartumNathan Campbell, Luke Strong, Xing Fang, et al.
Placenta|February 11, 2026
Tumor necrosis factor alpha inhibition improves fetal growth in a rat model of preeclampsiaLaura E Coats, Nathan Campbell, Dylan Solise, et al.
American Journal of Reproductive Immunology (New York, N.Y. : 1989)|April 12, 2024
The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancyJames P Hogg, Nathan Campbell, Evangeline Deer, et al.
American Journal of Physiology. Renal Physiology|November 16, 2020
CD4+ T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemiaEvangeline Deer, Kristin E Reeve, Lorena Amaral, et al.
Hypertension (Dallas, Tex. : 1979)|March 21, 2018
AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic RatsMark W Cunningham, Javier Castillo, Tarek Ibrahim, et al.
Journal of the American Heart Association|February 14, 2024
AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring HealthNathan Campbell, Evangeline Deer, Dylan Solise, et al.
Pageof 4

Showing results (11-20 of 40) with videos related to

Sort By:
Pageof 4
Hypertension in Pregnancy|May 3, 2026
Placental CD4⁺ T cells from women with gestational diabetes recapitulate disease features in a pregnant rat model, improved by metformin or mitotempoDanielle Frieson, Jean Vel, Tyler Johnson, et al.
Cells|November 27, 2021
Progesterone Induced Blocking Factor Reduces Hypertension and Placental Mitochondrial Dysfunction in Response to sFlt-1 during PregnancyEvangeline Deer, Jalisa Jones, Denise C Cornelius, et al.
Pregnancy Hypertension|March 7, 2021
The role of tumor necrosis factor in triggering activation of natural killer cell, multi-organ mitochondrial dysfunction and hypertension during pregnancyAswathi Jayaram, Evangeline Deer, Lorena M Amaral, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|October 28, 2016
Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancyDenise C Cornelius, Lorena M Amaral, Kedra Wallace, et al.
International Journal of Cerebrovascular Disease and Stroke|October 30, 2023
AT1-AA Infusion during Pregnancy Impairs CBF Autoregulation PostpartumNathan Campbell, Luke Strong, Xing Fang, et al.
Placenta|February 11, 2026
Tumor necrosis factor alpha inhibition improves fetal growth in a rat model of preeclampsiaLaura E Coats, Nathan Campbell, Dylan Solise, et al.
American Journal of Reproductive Immunology (New York, N.Y. : 1989)|April 12, 2024
The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancyJames P Hogg, Nathan Campbell, Evangeline Deer, et al.
American Journal of Physiology. Renal Physiology|November 16, 2020
CD4+ T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemiaEvangeline Deer, Kristin E Reeve, Lorena Amaral, et al.
Hypertension (Dallas, Tex. : 1979)|March 21, 2018
AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic RatsMark W Cunningham, Javier Castillo, Tarek Ibrahim, et al.
Journal of the American Heart Association|February 14, 2024
AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring HealthNathan Campbell, Evangeline Deer, Dylan Solise, et al.
Pageof 4