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International Journal of Proteomics|November 16, 2011
p73-Binding Partners and Their Functional SignificanceToshinori Ozaki, Natsumi Kubo, Akira Nakagawara
Biochemical and Biophysical Research Communications|June 16, 2009
Acetylation status of E2F-1 has an important role in the regulation of E2F-1-mediated transactivation of tumor suppressor p73Toshinori Ozaki, Rintaro Okoshi, Meixiang Sang, et al.
Biochemical and Biophysical Research Communications|July 7, 2009
Deregulated expression of E2F1 promotes proteolytic degradation of tumor suppressor p73 and inhibits its transcriptional activityToshinori Ozaki, Rintaro Okoshi, Sayaka Ono, et al.
The Journal of Biological Chemistry|November 14, 2012
Runt-related transcription factor 1 (RUNX1) stimulates tumor suppressor p53 protein in response to DNA damage through complex formation and acetylationDan Wu, Toshinori Ozaki, Yukari Yoshihara, et al.
Biochemical and Biophysical Research Communications|November 25, 2010
MDM2 promotes the proteasomal degradation of p73 through the interaction with Itch in HeLa cellsNatsumi Kubo, Rintaro Okoshi, Kumiko Nakashima, et al.
Biochemical and Biophysical Research Communications|December 25, 2012
Co-chaperon DnaJC7/TPR2 enhances p53 stability and activity through blocking the complex formation between p53 and MDM2Natsumi Kubo, Dan Wu, Yukari Yoshihara, et al.
Biochemical and Biophysical Research Communications|February 8, 2011
CREB represses p53-dependent transactivation of MDM2 through the complex formation with p53 and contributes to p53-mediated apoptosis in response to glucose deprivationRintaro Okoshi, Natsumi Kubo, Kumiko Nakashima, et al.
Biochemical and Biophysical Research Communications|April 7, 2012
Inhibitory role of E2F-1 in the regulation of tumor suppressor p53 during DNA damage responseYukari Yoshihara, Dan Wu, Natsumi Kubo, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|November 26, 2009
Transcriptional regulation of tumor suppressor p53 by cAMP-responsive element-binding protein/AMP-activated protein kinase complex in response to glucose deprivationRintaro Okoshi, Kiyohiro Ando, Yusuke Suenaga, et al.
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