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Bio-Protocol|November 11, 2024
Accurate Measurement of Cell Number-Normalized Differential Gene Expression in Cells Treated With Retinoic AcidNina Weichert-Leahey, Mark W Zimmerman, Alla Berezovskaya, et al.Cancer Discovery|December 30, 2017
MYC Drives a Subset of High-Risk Pediatric Neuroblastomas and Is Activated through Mechanisms Including Enhancer Hijacking and Focal Enhancer AmplificationMark W Zimmerman, Yu Liu, Shuning He, et al.The Journal of Clinical Investigation|May 15, 2023
Genetic predisposition to neuroblastoma results from a regulatory polymorphism that promotes the adrenergic cell stateNina Weichert-Leahey, Hui Shi, Ting Tao, et al.Biorxiv : the Preprint Server for Biology|March 13, 2023
Genetic Predisposition to Neuroblastoma Results from a Regulatory Polymorphism that Promotes the Adrenergic Cell StateNina Weichert-Leahey, Hui Shi, Ting Tao, et al.Nature Communications|December 11, 2019
ASCL1 is a MYCN- and LMO1-dependent member of the adrenergic neuroblastoma core regulatory circuitryLu Wang, Tze King Tan, Adam D Durbin, et al.Biorxiv : the Preprint Server for Biology|July 9, 2025
KAT6A/B inhibition synergizes with retinoic acid and enhances the efficacy of GD2-targeted immunotherapy in neuroblastomaNina Weichert-Leahey, Alla Berezovskaya, Mark W Zimmerman, et al.Nature Communications|June 2, 2017
Corrigendum: Small genomic insertions form enhancers that misregulate oncogenesBrian J Abraham, Denes Hnisz, Abraham S Weintraub, et al.Nature Communications|February 10, 2017
Small genomic insertions form enhancers that misregulate oncogenesBrian J Abraham, Denes Hnisz, Abraham S Weintraub, et al.Nature Genetics|August 22, 2018
Selective gene dependencies in MYCN-amplified neuroblastoma include the core transcriptional regulatory circuitryAdam D Durbin, Mark W Zimmerman, Neekesh V Dharia, et al.Blood|May 7, 2025
ANKRD26-related Thrombocytopenia 2 with a Baseline Increase in Blasts: Implications for Clinical SurveillanceLara Wahlster, Lucy A Godley, Jason X Cheng, et al.Pageof 2