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Nagoya Journal of Medical Science|September 21, 2011
Study profile on baseline survey of Daiko Study in the Japan Multi-Institutional Collaborative Cohort Study (J-MICC Study)Emi Morita, Nobuyuki Hamajima, Asahi Hishida, et al.
Scientific Reports|June 19, 2013
Common dysfunctional variants in ABCG2 are a major cause of early-onset goutHirotaka Matsuo, Kimiyoshi Ichida, Tappei Takada, et al.
British Journal of Haematology|February 14, 2002
Efficacy of granulocyte colony-stimulating factor in the treatment of acute myelogenous leukaemia: a multicentre randomized studyKensuke Usuki, Akio Urabe, Toru Masaoka, et al.
Science Translational Medicine|April 7, 2010
Common defects of ABCG2, a high-capacity urate exporter, cause gout: a function-based genetic analysis in a Japanese populationHirotaka Matsuo, Tappei Takada, Kimiyoshi Ichida, et al.
Annals of the Rheumatic Diseases|February 4, 2015
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Ken Yamamoto, Hirofumi Nakaoka, et al.
Journal of Epidemiology|September 23, 2020
Study Profile of the Japan Multi-institutional Collaborative Cohort (J-MICC) StudyKenji Takeuchi, Mariko Naito, Sayo Kawai, et al.
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