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P A Antinozzi

Showing results (1-10 of 9) with videos related to

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IUBMB Life|November 22, 2000
Modulation of glutamate generation in mitochondria affects hormone secretion in INS-1E beta cellsP Maechler, P A Antinozzi, C B Wollheim
Diabetes|July 27, 2001
Glycerol-stimulated proinsulin biosynthesis in isolated pancreatic rat islets via adenoviral-induced expression of glycerol kinase is mediated via mitochondrial metabolismR H Skelly, B Wicksteed, P A Antinozzi, et al.
Annual Review of Nutrition|August 17, 1999
Metabolic engineering with recombinant adenovirusesP A Antinozzi, H K Berman, R M O'Doherty, et al.
The Journal of Biological Chemistry|July 25, 1997
Engineering of glycerol-stimulated insulin secretion in islet beta cells. Differential metabolic fates of glucose and glycerol provide insight into mechanisms of stimulus-secretion couplingR J Noel, P A Antinozzi, J D McGarry, et al.
The EMBO Journal|August 16, 2000
Molecular targets of a human HNF1 alpha mutation responsible for pancreatic beta-cell dysfunctionH Wang, P A Antinozzi, K A Hagenfeldt, et al.
The Journal of Biological Chemistry|September 1, 2000
Hepatocyte nuclear factor 4alpha regulates the expression of pancreatic beta -cell genes implicated in glucose metabolism and nutrient-induced insulin secretionH Wang, P Maechler, P A Antinozzi, et al.
The Journal of Biological Chemistry|June 20, 1998
Molecular or pharmacologic perturbation of the link between glucose and lipid metabolism is without effect on glucose-stimulated insulin secretion. A re-evaluation of the long-chain acyl-CoA hypothesisP A Antinozzi, L Segall, M Prentki, et al.
Diabetologia|March 10, 2005
Suppression of Pdx-1 perturbs proinsulin processing, insulin secretion and GLP-1 signalling in INS-1 cellsH Wang, M Iezzi, S Theander, et al.
The Journal of Biological Chemistry|December 13, 2000
Overexpression of a modified human malonyl-CoA decarboxylase blocks the glucose-induced increase in malonyl-CoA level but has no impact on insulin secretion in INS-1-derived (832/13) beta-cellsH Mulder, D Lu, J Finley, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
IUBMB Life|November 22, 2000
Modulation of glutamate generation in mitochondria affects hormone secretion in INS-1E beta cellsP Maechler, P A Antinozzi, C B Wollheim
Diabetes|July 27, 2001
Glycerol-stimulated proinsulin biosynthesis in isolated pancreatic rat islets via adenoviral-induced expression of glycerol kinase is mediated via mitochondrial metabolismR H Skelly, B Wicksteed, P A Antinozzi, et al.
Annual Review of Nutrition|August 17, 1999
Metabolic engineering with recombinant adenovirusesP A Antinozzi, H K Berman, R M O'Doherty, et al.
The Journal of Biological Chemistry|July 25, 1997
Engineering of glycerol-stimulated insulin secretion in islet beta cells. Differential metabolic fates of glucose and glycerol provide insight into mechanisms of stimulus-secretion couplingR J Noel, P A Antinozzi, J D McGarry, et al.
The EMBO Journal|August 16, 2000
Molecular targets of a human HNF1 alpha mutation responsible for pancreatic beta-cell dysfunctionH Wang, P A Antinozzi, K A Hagenfeldt, et al.
The Journal of Biological Chemistry|September 1, 2000
Hepatocyte nuclear factor 4alpha regulates the expression of pancreatic beta -cell genes implicated in glucose metabolism and nutrient-induced insulin secretionH Wang, P Maechler, P A Antinozzi, et al.
The Journal of Biological Chemistry|June 20, 1998
Molecular or pharmacologic perturbation of the link between glucose and lipid metabolism is without effect on glucose-stimulated insulin secretion. A re-evaluation of the long-chain acyl-CoA hypothesisP A Antinozzi, L Segall, M Prentki, et al.
Diabetologia|March 10, 2005
Suppression of Pdx-1 perturbs proinsulin processing, insulin secretion and GLP-1 signalling in INS-1 cellsH Wang, M Iezzi, S Theander, et al.
The Journal of Biological Chemistry|December 13, 2000
Overexpression of a modified human malonyl-CoA decarboxylase blocks the glucose-induced increase in malonyl-CoA level but has no impact on insulin secretion in INS-1-derived (832/13) beta-cellsH Mulder, D Lu, J Finley, et al.
Pageof 1