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Sub-Cellular Biochemistry|January 16, 2008
Calcium and cell death: the mitochondrial connectionP Bernardi, A RasolaCell Death and Differentiation|January 10, 2004
Apoptosis to necrosis switching downstream of apoptosome formation requires inhibition of both glycolysis and oxidative phosphorylation in a BCL-X(L)- and PKB/AKT-independent fashionD Gramaglia, A Gentile, M Battaglia, et al.Cell Death & Disease|October 19, 2013
Myotonic dystrophy protein kinase (DMPK) prevents ROS-induced cell death by assembling a hexokinase II-Src complex on the mitochondrial surfaceB Pantic, E Trevisan, A Citta, et al.Cell Death & Disease|December 14, 2012
Chemotherapeutic induction of mitochondrial oxidative stress activates GSK-3α/β and Bax, leading to permeability transition pore opening and tumor cell deathF Chiara, A Gambalunga, M Sciacovelli, et al.Cytometry|October 9, 2001
A flow cytometry assay simultaneously detects independent apoptotic parametersA Rasola, M GeunaBiochimica Et Biophysica Acta|August 31, 1984
Modulation of Ca2+ efflux and rebounding Ca2+ transport in rat liver mitochondriaP BernardiThe Journal of Biological Chemistry|May 5, 1992
Modulation of the mitochondrial cyclosporin A-sensitive permeability transition pore by the proton electrochemical gradient. Evidence that the pore can be opened by membrane depolarizationP BernardiPhysiological Reviews|October 3, 1999
Mitochondrial transport of cations: channels, exchangers, and permeability transitionP BernardiBiochimica Et Biophysica Acta|July 18, 1996
The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell deathP BernardiPageof 20