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The Journal of Clinical Investigation|June 1, 1990
Bacterial phospholipid hydrolysis enhances the destruction of Escherichia coli ingested by rabbit neutrophils. Role of cellular and extracellular phospholipasesG C Wright, J Weiss, K S Kim, et al.
Biochimica Et Biophysica Acta|August 15, 1987
Relation between binding and the action of phospholipases A2 on Escherichia coli exposed to the bactericidal/permeability-increasing protein of neutrophilsS Forst, J Weiss, J M Maraganore, et al.
Journal of Lipid Research|July 1, 1980
The action of human and rabbit serum phospholipase A2 on Escherichia coli phospholipidsL Kaplan-Harris, J Weiss, C Mooney, et al.
Biochemistry|July 29, 1986
Amino acid sequence of a basic Agkistrodon halys blomhoffii phospholipase A2. Possible role of NH2-terminal lysines in action on phospholipids of Escherichia coliS Forst, J Weiss, P Blackburn, et al.
The Journal of Biological Chemistry|December 14, 1999
Deacylation of lipopolysaccharide in whole Escherichia coli during destruction by cellular and extracellular components of a rabbit peritoneal inflammatory exudateS S Katz, Y Weinrauch, R S Munford, et al.
The Journal of Biological Chemistry|July 25, 1997
Lipopolysaccharide (LPS)-binding proteins BPI and LBP form different types of complexes with LPSP S Tobias, K Soldau, N M Iovine, et al.
The Journal of Clinical Investigation|August 1, 1994
Individual and synergistic effects of rabbit granulocyte proteins on Escherichia coliO Levy, C E Ooi, J Weiss, et al.
Infection and Immunity|September 1, 1996
Potent CD14-mediated signalling of human leukocytes by Escherichia coli can be mediated by interaction of whole bacteria and host cells without extensive prior release of endotoxinS S Katz, K Chen, S Chen, et al.
Biochemistry|December 30, 1986
Structural and functional properties of a phospholipase A2 purified from an inflammatory exudateS Forst, J Weiss, P Elsbach, et al.
The Journal of Clinical Investigation|September 1, 1992
Human bactericidal/permeability-increasing protein and a recombinant NH2-terminal fragment cause killing of serum-resistant gram-negative bacteria in whole blood and inhibit tumor necrosis factor release induced by the bacteriaJ Weiss, P Elsbach, C Shu, et al.
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