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The New England Journal of Medicine|February 4, 1982
An association between neonatal severe primary hyperparathyroidism and familial hypocalciuric hypercalcemia in three kindredsS J Marx, M F Attie, A M Spiegel, et al.
Cancer Research|December 4, 2010
The tumor suppressor protein menin inhibits AKT activation by regulating its cellular localizationYan Wang, Atsushi Ozawa, Shadia Zaman, et al.
The Journal of Clinical Endocrinology and Metabolism|January 1, 1982
Human chorionic gonadotropin subunit measurement in primary hyperparathyroidismJ L Stock, B D Weintraub, S W Rosen, et al.
Surgery|June 1, 1978
Reoperative parathyroid surgery for persistent hyperparathyroidismM F Brennan, J L Doppman, S J Marx, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 22, 2015
Galactose metabolic genes in yeast respond to a ratio of galactose and glucoseRenan Escalante-Chong, Yonatan Savir, Sean M Carroll, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|November 5, 2002
Role of preoperative localization and intraoperative localization maneuvers including intraoperative PTH assay determination for patients with persistent or recurrent hyperparathyroidismH Richard Alexander, Clara C Chen, Thomas Shawker, et al.
Oncogene|September 26, 2003
Menin, a tumor suppressor, associates with nonmuscle myosin II-A heavy chainVictor H Obungu, A Lee Burns, Sunita K Agarwal, et al.
The Journal of Clinical Endocrinology and Metabolism|January 13, 2004
Familial isolated hyperparathyroidism is rarely caused by germline mutation in HRPT2, the gene for the hyperparathyroidism-jaw tumor syndromeWilliam F Simonds, Christiane M Robbins, Sunita K Agarwal, et al.
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