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Neuromolecular Medicine|June 10, 2019
Sideroflexin 3 is a Mitochondrial Protein Enriched in NeuronsAileen Rivell, Ronald S Petralia, Ya-Xian Wang, et al.
Antioxidants & Redox Signaling|May 8, 2010
Cellular stress responses, the hormesis paradigm, and vitagenes: novel targets for therapeutic intervention in neurodegenerative disordersVittorio Calabrese, Carolin Cornelius, Albena T Dinkova-Kostova, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 1, 1992
beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicityM P Mattson, B Cheng, D Davis, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|November 18, 2020
Age-related impairment of cerebral blood flow response to K<sub>ATP</sub> channel opener in Alzheimer's disease mice with presenilin-1 mutationDong Liu, Ismayil Ahmet, Brandon Griess, et al.
Trends in Neurosciences|October 22, 2010
When neurogenesis encounters aging and diseaseOrly Lazarov, Mark P Mattson, Daniel A Peterson, et al.
The Journal of Comparative Neurology|July 24, 2010
Clathrin assembly proteins AP180 and CALM in the embryonic rat brainCatherine M Schwartz, Aiwu Cheng, Mohamed R Mughal, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 26, 1995
Tumor necrosis factors alpha and beta protect neurons against amyloid beta-peptide toxicity: evidence for involvement of a kappa B-binding factor and attenuation of peroxide and Ca2+ accumulationS W Barger, D Hörster, K Furukawa, et al.
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