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The Yale Journal of Biology and Medicine|January 1, 1982
Does ATP cross the cell plasma membraneI H ChaudryAnnals of the New York Academy of Sciences|January 1, 1990
Use of ATP following shock and ischemiaI H ChaudryThe American Journal of Physiology|August 1, 1983
Cellular mechanisms in shock and ischemia and their correctionI H ChaudryThe American Journal of Emergency Medicine|January 1, 1984
Cellular energetics and ATP-MgCl2 therapy in sepsisI H ChaudryProgress in Clinical and Biological Research|January 1, 1989
ATP-MgCl2 and liver blood flow following shock and ischemiaI H ChaudryCritical Care Medicine|June 8, 2001
Glycine attenuates hepatocellular depression during early sepsis and reduces sepsis-induced mortalityS Yang, D J Koo, I H Chaudry, et al.The Journal of Trauma|March 1, 1992
Hepatocellular dysfunction persists during early sepsis despite increased volume of crystalloid resuscitationP Wang, Z F Ba, A Ayala, et al.Archives of Surgery (Chicago, Ill. : 1960)|February 11, 2000
Plasma alpha-glutathione S-transferase: a sensitive indicator of hepatocellular damage during polymicrobial sepsisD J Koo, M Zhou, I H Chaudry, et al.The Journal of Surgical Research|February 13, 2001
The role of adrenomedullin in producing differential hemodynamic responses during sepsisD J Koo, M Zhou, I H Chaudry, et al.The Journal of Surgical Research|June 1, 1992
Sepsis induces an early increased spontaneous release of hepatocellular stimulatory factor (interleukin-6) by Kupffer cells in both endotoxin tolerant and intolerant miceA Ayala, M M Perrin, P Wang, et al.Pageof 498