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European Journal of Histochemistry : EJH|October 22, 2019
Digital PCR - Methods and ProtocolsPaolo CatarsiOncogene|July 12, 2005
Synergistic inhibition of human neuroblastoma-related angiogenesis by vinblastine and rapamycinDanilo Marimpietri, Beatrice Nico, Angelo Vacca, et al.Physiological Genomics|October 14, 2004
Polymorphisms in the osteopontin promoter affect its transcriptional activityFrancesca Giacopelli, Renato Marciano, Angela Pistorio, et al.Experimental Hematology|October 16, 2022
Cells coexpressing both myeloid and endothelial markers are detectable in the spleen and bone marrow of patients with primary myelofibrosisRita Campanelli, Carlotta Abbà, Adriana Carolei, et al.Current Clinical Pharmacology|September 11, 2008
Cyclosporin and organ specific toxicity: clinical aspects, pharmacogenetics and perspectivesAlberto Magnasco, Andrea Rossi, Paolo Catarsi, et al.Plos One|January 25, 2015
JAK2 exon 14 skipping in patients with primary myelofibrosis: a minor splice variant modulated by the JAK2-V617F allele burdenPaolo Catarsi, Vittorio Rosti, Giacomo Morreale, et al.Plos One|August 2, 2019
Constitutive STAT5 phosphorylation in CD34+ cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severityCarlotta Abbà, Rita Campanelli, Paolo Catarsi, et al.Human Molecular Genetics|July 9, 2005
Angiotensin-converting enzyme (ACE) haplotypes and cyclosporine A (CsA) response: a model of the complex relationship between ACE quantitative trait locus and pathological phenotypesPaolo Catarsi, Roberto Ravazzolo, Francesco Emma, et al.Genes|August 27, 2021
Clinical Relevance of VEGFA (rs3025039) +936 C>T Polymorphism in Primary Myelofibrosis: Susceptibility, Clinical Co-Variates, and OutcomesLaura Villani, Adriana Carolei, Vittorio Rosti, et al.Plos One|April 5, 2013
JAK2 V617F genotype is a strong determinant of blast transformation in primary myelofibrosisGiovanni Barosi, Valentina Poletto, Margherita Massa, et al.Pageof 4