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Neuron|December 10, 2020
Mitigating Cognitive Deficits in Down Syndrome by Managing Microglia ActivationPatricia R Shaw, Tarik F HaydarDisease Models & Mechanisms|August 21, 2020
Longitudinal neuroanatomical and behavioral analyses show phenotypic drift and variability in the Ts65Dn mouse model of Down syndromePatricia R Shaw, Jenny A Klein, Nadine M Aziz, et al.Mental Retardation and Developmental Disabilities Research Reviews|October 22, 2005
Advanced microscopic imaging methods to investigate cortical development and the etiology of mental retardationTarik F HaydarAmerican Journal on Intellectual and Developmental Disabilities|February 15, 2020
Neurogenesis, Myelination, and Circuitry: The Case for a Distributed Therapeutic Regimen in Down SyndromeTarik F HaydarBiorxiv : the Preprint Server for Biology|December 9, 2024
MuMu: a sample multiplexing protocol for droplet-based simultaneous single nuclei RNA- and ATAC-seq systemsZhen Li, Tarik F HaydarFrontiers in Neuroscience|June 11, 2026
Progenitor diversity during formation of the mammalian neocortexZeynep Atak, Tarik F HaydarFrontiers in Cellular Neuroscience|August 1, 2022
Neurodevelopment in Down syndrome: Concordance in humans and modelsJenny A Klein, Tarik F HaydarThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 22, 2013
Multiplex genetic fate mapping reveals a novel route of neocortical neurogenesis, which is altered in the Ts65Dn mouse model of Down syndromeWilliam A Tyler, Tarik F HaydarCerebral Cortex (New York, N.Y. : 1991)|November 26, 2011
The (not necessarily) convoluted role of basal radial glia in cortical neurogenesisRobert F Hevner, Tarik F HaydarTrends in Neurosciences|December 16, 2011
Trisomy 21 and early brain developmentTarik F Haydar, Roger H ReevesPageof 6