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The Plant Cell|April 11, 2022
PAMP-triggered genetic reprogramming involves widespread alternative transcription initiation and an immediate transcription factor waveAxel Thieffry, Diego López-Márquez, Jette Bornholdt, et al.
Elife|September 30, 2021
The YTHDF proteins ECT2 and ECT3 bind largely overlapping target sets and influence target mRNA abundance, not alternative polyadenylationLaura Arribas-Hernández, Sarah Rennie, Michael Schon, et al.
EMBO Reports|February 28, 2020
Organismal benefits of transcription speed control at gene boundariesXueyuan Leng, Maxim Ivanov, Peter Kindgren, et al.
Development (Cambridge, England)|July 3, 2020
Recurrent requirement for the m<sup>6</sup>A-ECT2/ECT3/ECT4 axis in the control of cell proliferation during plant organogenesisLaura Arribas-Hernández, Sara Simonini, Mathias Henning Hansen, et al.
Plos Genetics|October 10, 2023
Insights into the conservation and diversification of the molecular functions of YTHDF proteinsDaniel Flores-Téllez, Mathias Due Tankmar, Sören von Bülow, et al.
Scientific Reports|December 29, 2019
Plasmonics of Diffused Silver Nanoparticles in Silver/Nitride Optical Thin FilmsYufeng Ye, Joel Y Y Loh, Andrew Flood, et al.
Plos Pathogens|October 16, 2010
Autoimmunity in Arabidopsis acd11 is mediated by epigenetic regulation of an immune receptorKristoffer Palma, Stephan Thorgrimsen, Frederikke Gro Malinovsky, et al.
The Plant Journal : for Cell and Molecular Biology|July 4, 2006
Arabidopsis MAP kinase 4 regulates salicylic acid- and jasmonic acid/ethylene-dependent responses via EDS1 and PAD4Peter Brodersen, Morten Petersen, Henrik Bjørn Nielsen, et al.
Genes & Development|February 19, 2002
Knockout of Arabidopsis accelerated-cell-death11 encoding a sphingosine transfer protein causes activation of programmed cell death and defensePeter Brodersen, Morten Petersen, Helen M Pike, et al.
The FEBS Journal|July 29, 2008
Human GLTP and mutant forms of ACD11 suppress cell death in the Arabidopsis acd11 mutantNikolaj H T Petersen, Lea V McKinney, Helen Pike, et al.
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