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Methods in Molecular Medicine|December 15, 2005
In vitro assays of substrate degradation induced by high-risk HPV E6 oncoproteinsMiranda Thomas, Lawrence BanksJournal of Virology|August 25, 2017
Human Papillomavirus 16 (HPV-16), HPV-18, and HPV-31 E6 Override the Normal Phosphoregulation of E6AP Enzymatic ActivityJayashree Thatte, Lawrence BanksJournal of Virology|October 31, 2014
PDZRN3/LNX3 is a novel target of human papillomavirus type 16 (HPV-16) and HPV-18 E6Miranda Thomas, Lawrence BanksJournal of Virology|November 2, 2012
SNX17 facilitates infection with diverse papillomavirus typesMartina Bergant, Lawrence BanksCurrent Opinion in Virology|October 16, 2021
The biology of papillomavirus PDZ associations: what do they offer papillomaviruses?Miranda Thomas, Lawrence BanksInternational Journal of Cancer|November 26, 2003
p53 polymorphic variants at codon 72 exert different effects on cell cycle progressionDavid Pim, Lawrence BanksThe FEBS Journal|October 4, 2017
Viral oncoproteins and ubiquitination: accessing a cellular toolbox for modifying protein functionMiranda Thomas, Lawrence BanksThe Journal of Biological Chemistry|August 7, 2003
Regulation of the discs large tumor suppressor by a phosphorylation-dependent interaction with the beta-TrCP ubiquitin ligase receptorFiamma Mantovani, Lawrence BanksViruses|October 26, 2021
The Not-So-Good, the Bad and the Ugly: HPV E5, E6 and E7 Oncoproteins in the Orchestration of CarcinogenesisOm Basukala, Lawrence BanksAPMIS : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica|June 18, 2010
Interaction of viral oncoproteins with cellular target molecules: infection with high-risk vs low-risk human papillomavirusesDavid Pim, Lawrence BanksPageof 25