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Plos Pathogens|May 23, 2019
The HPV-18 E7 CKII phospho acceptor site is required for maintaining the transformed phenotype of cervical tumour-derived cellsOm Basukala, Suruchi Mittal, Paola Massimi, et al.Journal of Cellular Biochemistry|April 23, 2002
Human papillomavirus-16 E7 protein inhibits the DNA interaction of the TATA binding transcription factorEdio Maldonado, María Eugenia Cabrejos, Lawrence Banks, et al.Virology Journal|June 4, 2008
Comparison of p53 and the PDZ domain containing protein MAGI-3 regulation by the E6 protein from high-risk human papillomavirusesJulia Ainsworth, Miranda Thomas, Lawrence Banks, et al.Tumour Virus Research|March 14, 2024
HPV16 E7 modulates the cell surface expression of MET and CD109 via the AP2 complexOscar Trejo-Cerro, Om Basukala, Michael P Myers, et al.Scientific Reports|January 18, 2018
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entryJustyna Broniarczyk, Nadja Ring, Paola Massimi, et al.Journal of Virology|May 27, 2026
Rethinking residual p53 function in HPV-positive cervical cancer cellsSannia Farrukh, Nezka Kavcic, Giulia Canarutto, et al.Gene|August 30, 2008
Cloning and functional analysis of the promoter region of the human Disc large geneAna Laura Cavatorta, Adriana A Giri, Lawrence Banks, et al.Journal of Virology|November 12, 2020
Human Papillomavirus 16 L2 Recruits both Retromer and Retriever Complexes during Retrograde Trafficking of the Viral Genome to the Cell NucleusDavid Pim, Justyna Broniarczyk, Abida Siddiqa, et al.Journal of Virology|January 12, 2018
Human Papillomavirus 16 Infection Induces VAP-Dependent Endosomal TubulationAbida Siddiqa, Paola Massimi, David Pim, et al.International Journal of Cancer|April 19, 2006
Human discs large and scrib are localized at the same regions in colon mucosa and changes in their expression patterns are correlated with loss of tissue architecture during malignant progressionDaniela Gardiol, Alberto Zacchi, Francesca Petrera, et al.Pageof 25