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EMBO Reports|July 4, 2009
The NALCN ion channel is activated by M3 muscarinic receptors in a pancreatic beta-cell lineLeigh Anne Swayne, Alexandre Mezghrani, Annie Varrault, et al.Idrugs : the Investigational Drugs Journal|June 29, 2010
Calcium channelopathies in inherited neurological disorders: relevance to drug screening for acquired channel disordersPhilippe Lory, Alexandre MezghraniFrontiers in Pharmacology|October 21, 2021
New Challenges Resulting From the Loss of Function of Nav1.4 in Neuromuscular DiseasesSophie Nicole, Philippe LoryThe Journal of Physiology|July 5, 2016
Rescuing cardiac automaticity in L-type Cav1.3 channelopathies and beyondPietro Mesirca, Isabelle Bidaud, Matteo E MangoniNature Communications|January 3, 2024
Selective blockade of Cav1.2 (α1C) versus Cav1.3 (α1D) L-type calcium channels by the black mamba toxin calciseptinePietro Mesirca, Jean Chemin, Christian Barrère, et al.Ebiomedicine|October 13, 2016
CACNA1H Mutations Are Associated With Different Forms of Primary AldosteronismGeorgios Daniil, Fabio L Fernandes-Rosa, Jean Chemin, et al.The Journal of Physiology|March 3, 2016
Genetic alteration of the metal/redox modulation of Cav3.2 T-type calcium channel reveals its role in neuronal excitabilityTiphaine Voisin, Emmanuel Bourinet, Philippe LoryEndocrinology|April 17, 2003
Pharmacological studies of thyrotropin-releasing hormone (TRH) receptors from Xenopus laevis: is xTRHR3 a TRH receptor?Xinping Lu, Isabelle Bidaud, Ali Ladram, et al.Pflugers Archiv : European Journal of Physiology|July 9, 2020
Neuronal Cav3 channelopathies: recent progress and perspectivesPhilippe Lory, Sophie Nicole, Arnaud MonteilPflugers Archiv : European Journal of Physiology|July 11, 2020
Correction to: Channelopathies of voltage-gated L-type Cav1.3/α1D and T-type Cav3.1/α1G Ca2+ channels in dysfunction of heart automaticityAngelo G Torrente, Pietro Mesirca, Isabelle Bidaud, et al.Pageof 12