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Nature|January 17, 2002
Mechanism for the learning deficits in a mouse model of neurofibromatosis type 1Rui M Costa, Nikolai B Federov, Jeff H Kogan, et al.Genes & Development|November 12, 2009
Dicer1 functions as a haploinsufficient tumor suppressorMadhu S Kumar, Ryan E Pester, Cindy Y Chen, et al.Nature Biotechnology|April 1, 2014
Genome editing with Cas9 in adult mice corrects a disease mutation and phenotypeHao Yin, Wen Xue, Sidi Chen, et al.Proceedings of the National Academy of Sciences of the United States of America|May 11, 2011
Selective killing of K-ras mutant cancer cells by small molecule inducers of oxidative stressAlice T Shaw, Monte M Winslow, Margaret Magendantz, et al.Nature|January 26, 2007
Restoration of p53 function leads to tumour regression in vivoAndrea Ventura, David G Kirsch, Margaret E McLaughlin, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|September 17, 2002
Inhibition of oncogenic K-ras signaling by aerosolized gene delivery in a mouse model of human lung cancerHo-Young Lee, Young-Ah Suh, Janet I Lee, et al.Nano Letters|March 14, 2018
Differences in Nanoparticle Uptake in Transplanted and Autochthonous Models of Pancreatic CancerZhimin Tao, Mandar Deepak Muzumdar, Alexandre Detappe, et al.Cell Reports|November 19, 2013
A reversible gene-targeting strategy identifies synthetic lethal interactions between MK2 and p53 in the DNA damage response in vivoSandra Morandell, H Christian Reinhardt, Ian G Cannell, et al.Cancer Research|April 22, 2010
Impaired SHP2-mediated extracellular signal-regulated kinase activation contributes to gefitinib sensitivity of lung cancer cells with epidermal growth factor receptor-activating mutationsMatthew J Lazzara, Keara Lane, Richard Chan, et al.Nature Communications|November 6, 2015
Recombinase-based conditional and reversible gene regulation via XTR allelesCamila Robles-Oteiza, Sarah Taylor, Travis Yates, et al.Pageof 25