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European Thyroid Journal|May 22, 2014
Major Haemorrhage during Vitamin K Antagonist Treatment: The Influence of Thyroid Hormone LevelsJan Debeij, Suzanne C Cannegieter, Bregje van Zaane, et al.Nature Reviews. Disease Primers|May 18, 2018
Pulmonary embolismMenno V Huisman, Stefano Barco, Suzanne C Cannegieter, et al.The Journal of Laboratory and Clinical Medicine|June 6, 2006
Expression of inflammation-related genes in endothelial cells is not directly affected by microparticles from preeclamptic patientsChristianne Anne Rachel Lok, Anita N Böing, Pieter H Reitsma, et al.Basic Research in Cardiology|October 14, 2003
Functional thrombomodulin deficiency causes enhanced thrombus growth in a murine model of carotid artery thrombosisJanine Dörffler-Melly, Martijn de Kruif, Lothar A Schwarte, et al.Plos One|December 10, 2016
Objectives and Design of BLEEDS: A Cohort Study to Identify New Risk Factors and Predictors for Major Bleeding during Treatment with Vitamin K AntagonistsNienke van Rein, Willem M Lijfering, Mettine H A Bos, et al.Human Mutation|April 26, 2008
PROS1 analysis in 87 pedigrees with hereditary protein S deficiency demonstrates striking genotype-phenotype associationsMin Ki Ten Kate, Mathieu Platteel, Rene Mulder, et al.Contemporary Topics in Laboratory Animal Science|June 8, 2002
Fentanyl-fluanisone-midazolam combination results in more stable hemodynamics than does urethane alpha-chloralose and 2,2,2-tribromoethanol in miceWilleke M C Jong, Coert J Zuurbier, Robbert J De Winter, et al.Blood|November 1, 2003
Thrombomodulin mutant mice with a strongly reduced capacity to generate activated protein C have an unaltered pulmonary immune response to respiratory pathogens and lipopolysaccharideAnita W Rijneveld, Sebastiaan Weijer, Sandrine Florquin, et al.Thrombosis and Haemostasis|August 28, 2020
High Soluble Thrombomodulin Is Associated with an Increased Risk of Major Bleeding during Treatment with Oral Anticoagulants: A Case-Cohort StudyMyrthe M A Toorop, Nienke van Rein, Suzanne C Cannegieter, et al.Journal of Clinical Immunology|May 22, 2003
Treatment with an anti-CD14 monoclonal antibody delays and inhibits lipopolysaccharide-induced gene expression in humans in vivoC Arnold Spek, Annelies Verbon, Hella Aberson, et al.Pageof 11