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Proceedings. Biological Sciences|July 22, 1996
Anti-CD4 therapy for AIDS suggested by mathematical modelsR J De Boer, C A BoucherJournal of Virology|November 9, 2000
Estimating relative fitness in viral competition experimentsA F Marée, W Keulen, C A Boucher, et al.Journal of Virology|January 1, 1997
Clinical data sets of human immunodeficiency virus type 1 reverse transcriptase-resistant mutants explained by a mathematical modelN I Stilianakis, C A Boucher, M D De Jong, et al.Proceedings of the National Academy of Sciences of the United States of America|May 28, 1996
Host-parasite dynamics and outgrowth of virus containing a single K70R amino acid change in reverse transcriptase are responsible for the loss of human immunodeficiency virus type 1 RNA load suppression by zidovudineM D de Jong, J Veenstra, N I Stilianakis, et al.The Netherlands Journal of Medicine|November 15, 2002
Mathematical models of human CD4+ T-cell population kineticsR J de BoerThe Netherlands Journal of Medicine|October 1, 1991
Recent developments in idiotypic network theoryR J De BoerImmunology Letters|August 1, 1989
Poor repertoire selection in symmetric idiotypic network modelsR J De BoerAIDS (London, England)|September 26, 1997
Overshoot of HIV-1 viraemia after early discontinuation of antiretroviral treatmentM D de Jong, R J de Boer, F de Wolf, et al.Blood|December 23, 1999
T-cell division in human immunodeficiency virus (HIV)-1 infection is mainly due to immune activation: a longitudinal analysis in patients before and during highly active antiretroviral therapy (HAART)M D Hazenberg, J W Stuart, S A Otto, et al.Journal of Acquired Immune Deficiency Syndromes (1999)|December 15, 2000
The dominant source of CD4+ and CD8+ T-cell activation in HIV infection is antigenic stimulationJ W Cohen Stuart, M D Hazebergh, D Hamann, et al.Pageof 31