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Carcinogenesis; a Comprehensive Survey|January 1, 1982
Suppression of natural tumor resistance by tumor promotersR Keller, R Keist, P Köppel, et al.Experimental Cell Biology|January 1, 1982
Tumor-promoting diterpene esters prevent macrophage activation and suppress macrophage tumoricidal capacityR Keller, R Keist, W Adolf, et al.Biochemical and Biophysical Research Communications|November 15, 1989
Abilities of activated macrophages to manifest tumoricidal activity and to generate reactive nitrogen intermediates: a comparative study in vitro and ex vivoR Keller, R KeistExperimental Cell Biology|January 1, 1982
Tumor-promoting phorbol esters induce macrophage differentiation from bone marrow precursorsR Keller, R KeistEuropean Journal of Immunology|November 1, 1987
Induction of interleukin 1 secretion and of tumoricidal activity in macrophages are not closely related phenomenaR Keller, R KeistCellular Immunology|September 1, 1986
Induction, maintenance, and reinduction of tumoricidal activity in bone-marrow-derived mononuclear phagocytes by macrophage-activating lymphokinesR Keller, R KeistEuropean Journal of Immunology|March 1, 1990
Lymphokines and bacteria, that induce tumoricidal activity, trigger a different secretory response in macrophagesR Keller, R Keist, K FreiScandinavian Journal of Immunology|September 1, 1995
Macrophage response to microbial pathogens: modulation of the expression of adhesion, CD14, and MHC class II molecules by viruses, bacteria, protozoa and fungiR Keller, R Keist, P JollerCellular Immunology|April 15, 1992
The interaction of macrophages and bacteria: Escherichia coli species, bacterial lipopolysaccharide, and lipid A differ in their ability to induce tumoricidal activity and the secretion of reactive nitrogen intermediates in macrophagesR Keller, R Gehri, R KeistInternational Journal of Cancer|January 15, 1986
Firm persistent binding between activated macrophages and tumor cells is not a prerequisite for the mediation of cytolysisR Keller, R Keist, P GroscurthPageof 105