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R Kew

Showing results (11-20 of 60) with videos related to

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Molecular Immunology|December 4, 2014
Enhanced recognition of plasma proteins in a non-native state by complement C3b. A possible clearance mechanism for damaged proteins in bloodMahalakshmi Ramadass, Berhane Ghebrehiwet, Richard R Kew
Clinical Immunology and Immunopathology|August 1, 1987
Characterization of the third component of complement (C3) after activation by cigarette smokeR R Kew, B Ghebrehiwet, A Janoff
Blood|October 1, 1995
Angiogenic factors stimulate mast-cell migrationB L Gruber, M J Marchese, R Kew
Advances in Biological Regulation|November 22, 2016
Sphingolipids in neutrophil function and inflammatory responses: Mechanisms and implications for intestinal immunity and inflammation in ulcerative colitisMel Pilar Espaillat, Richard R Kew, Lina M Obeid
Journal of Immunology (Baltimore, Md. : 1950)|June 15, 1994
Transforming growth factor-beta 1 mediates mast cell chemotaxisB L Gruber, M J Marchese, R R Kew
Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1995
Co-chemotactic effect of Gc-globulin (vitamin D binding protein) for C5a. Transient conversion into an active co-chemotaxin by neutrophilsR R Kew, J A Fisher, R O Webster
Journal of Leukocyte Biology|July 1, 1995
Binding of Gc globulin (vitamin D binding protein) to C5a or C5a des Arg is not necessary for co-chemotactic activityR R Kew, K W Mollison, R O Webster
Clinical Immunology and Immunopathology|July 1, 1998
Smokeless tobacco extracts activate complement in vitro: a potential pathogenic mechanism for initiating inflammation of the oral mucosaT Chang, S Chowdhry, P Budhu, et al.
The Journal of Laboratory and Clinical Medicine|March 1, 1990
Human C-reactive protein inhibits neutrophil chemotaxis in vitro: possible implications for the adult respiratory distress syndromeR R Kew, T M Hyers, R O Webster
Molecular Immunology|September 6, 2005
Chemotaxis of human monocyte-derived dendritic cells to complement component C1q is mediated by the receptors gC1qR and cC1qRZsuzsa Vegh, Richard R Kew, Barry L Gruber, et al.
Pageof 6

Showing results (11-20 of 60) with videos related to

Sort By:
Pageof 6
Molecular Immunology|December 4, 2014
Enhanced recognition of plasma proteins in a non-native state by complement C3b. A possible clearance mechanism for damaged proteins in bloodMahalakshmi Ramadass, Berhane Ghebrehiwet, Richard R Kew
Clinical Immunology and Immunopathology|August 1, 1987
Characterization of the third component of complement (C3) after activation by cigarette smokeR R Kew, B Ghebrehiwet, A Janoff
Blood|October 1, 1995
Angiogenic factors stimulate mast-cell migrationB L Gruber, M J Marchese, R Kew
Advances in Biological Regulation|November 22, 2016
Sphingolipids in neutrophil function and inflammatory responses: Mechanisms and implications for intestinal immunity and inflammation in ulcerative colitisMel Pilar Espaillat, Richard R Kew, Lina M Obeid
Journal of Immunology (Baltimore, Md. : 1950)|June 15, 1994
Transforming growth factor-beta 1 mediates mast cell chemotaxisB L Gruber, M J Marchese, R R Kew
Journal of Immunology (Baltimore, Md. : 1950)|December 1, 1995
Co-chemotactic effect of Gc-globulin (vitamin D binding protein) for C5a. Transient conversion into an active co-chemotaxin by neutrophilsR R Kew, J A Fisher, R O Webster
Journal of Leukocyte Biology|July 1, 1995
Binding of Gc globulin (vitamin D binding protein) to C5a or C5a des Arg is not necessary for co-chemotactic activityR R Kew, K W Mollison, R O Webster
Clinical Immunology and Immunopathology|July 1, 1998
Smokeless tobacco extracts activate complement in vitro: a potential pathogenic mechanism for initiating inflammation of the oral mucosaT Chang, S Chowdhry, P Budhu, et al.
The Journal of Laboratory and Clinical Medicine|March 1, 1990
Human C-reactive protein inhibits neutrophil chemotaxis in vitro: possible implications for the adult respiratory distress syndromeR R Kew, T M Hyers, R O Webster
Molecular Immunology|September 6, 2005
Chemotaxis of human monocyte-derived dendritic cells to complement component C1q is mediated by the receptors gC1qR and cC1qRZsuzsa Vegh, Richard R Kew, Barry L Gruber, et al.
Pageof 6