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European Journal of Endocrinology|October 7, 2000
The highly malignant phenotype of anaplastic thyroid carcinoma cell lines is recessiveM L Martelli, M G Miano, C Battaglia, et al.Oncogene|September 21, 1995
RET/PTC oncogene activation is an early event in thyroid carcinogenesisG Viglietto, G Chiappetta, F J Martinez-Tello, et al.Oncogene|April 6, 1995
The expression of the high mobility group HMGI (Y) proteins correlates with the malignant phenotype of human thyroid neoplasiasG Chiappetta, A Bandiera, M T Berlingieri, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|January 1, 1996
Block of c-myc expression by antisense oligonucleotides inhibits proliferation of human thyroid carcinoma cell linesJ Cerutti, F Trapasso, C Battaglia, et al.Cancer Research|August 5, 2000
Tyrosine 1062 of RET-MEN2A mediates activation of Akt (protein kinase B) and mitogen-activated protein kinase pathways leading to PC12 cell survivalG De Vita, R M Melillo, F Carlomagno, et al.Endocrinology|July 29, 1998
Glial cell line-derived neurotrophic factor differentially stimulates ret mutants associated with the multiple endocrine neoplasia type 2 syndromes and Hirschsprung's diseaseF Carlomagno, R M Melillo, R Visconti, et al.Oncogene|April 21, 2001
The RFG oligomerization domain mediates kinase activation and re-localization of the RET/PTC3 oncoprotein to the plasma membraneC Monaco, R Visconti, M V Barone, et al.Oncogene|December 5, 1996
High level expression of the HMGI (Y) gene during embryonic developmentG Chiappetta, V Avantaggiato, R Visconti, et al.Oncogene|November 19, 1997
Expression of the neoplastic phenotype by human thyroid carcinoma cell lines requires NFkappaB p65 protein expressionR Visconti, J Cerutti, S Battista, et al.Cancer Research|August 5, 2000
Akt/protein kinase B promotes survival and hormone-independent proliferation of thyroid cells in the absence of dedifferentiating and transforming effectsG De Vita, M T Berlingieri, R Visconti, et al.Pageof 4