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Microbial Pathogenesis|March 8, 2005
Comparison of wild type with recombinant Clostridium difficile toxin ARalf Gerhard, Silke Burger, Helma Tatge, et al.Biochemical and Biophysical Research Communications|August 2, 2003
Expression of recombinant Clostridium difficile toxin A using the Bacillus megaterium systemSilke Burger, Helma Tatge, Fred Hofmann, et al.The Journal of Biological Chemistry|November 9, 2004
Clostridium difficile toxin A induces expression of the stress-induced early gene product RhoBRalf Gerhard, Helma Tatge, Harald Genth, et al.FEBS Letters|May 30, 2006
Cellular stability of Rho-GTPases glucosylated by Clostridium difficile toxin BHarald Genth, Johannes Huelsenbeck, Birgit Hartmann, et al.Infection and Immunity|September 22, 2006
Application of mutated Clostridium difficile toxin A for determination of glucosyltransferase-dependent effectsMatthias Teichert, Helma Tatge, Janett Schoentaube, et al.Cellular Microbiology|August 28, 2009
Serine-71 phosphorylation of Rac1/Cdc42 diminishes the pathogenic effect of Clostridium difficile toxin AJanett Schoentaube, Alexandra Olling, Helma Tatge, et al.Biochemistry|February 10, 2009
Killing of rat basophilic leukemia cells by lethal toxin from Clostridium sordellii: critical role of phosphatidylinositide 3'-OH kinase/Akt signalingStefanie C Dreger, Florian Schulz, Johannes Huelsenbeck, et al.Journal of Medical Microbiology|May 16, 2008
Glucosylation of Rho GTPases by Clostridium difficile toxin A triggers apoptosis in intestinal epithelial cellsRalf Gerhard, Stefanie Nottrott, Janett Schoentaube, et al.Plos One|March 30, 2011
The repetitive oligopeptide sequences modulate cytopathic potency but are not crucial for cellular uptake of Clostridium difficile toxin AAlexandra Olling, Sebastian Goy, Florian Hoffmann, et al.The Journal of Biological Chemistry|March 29, 2007
Neogenin-RGMa signaling at the growth cone is bone morphogenetic protein-independent and involves RhoA, ROCK, and PKCSabine Conrad, Harald Genth, Fred Hofmann, et al.Pageof 17