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Methods in Molecular Biology (Clifton, N.J.)|June 1, 2018
Mitochondrial Permeability Transition Pore and Calcium HandlingRandi J Parks, Elizabeth Murphy, Julia C LiuAdvances in Experimental Medicine and Biology|May 29, 2017
The In Vivo Biology of the Mitochondrial Calcium UniporterJulia C Liu, Randi J Parks, Jie Liu, et al.Cardiovascular Research|February 4, 2017
Sex Differences in Metabolic CardiomyopathyElizabeth Murphy, Georgios Amanakis, Natasha Fillmore, et al.Cardiovascular Research|August 20, 2022
Mitochondrial calcium and reactive oxygen species in cardiovascular diseaseElizabeth Murphy, Julia C LiuJournal of Molecular and Cellular Cardiology|June 10, 2015
Assessment of cardiac function in mice lacking the mitochondrial calcium uniporterKira M Holmström, Xin Pan, Julia C Liu, et al.Cell Reports|August 2, 2016
MICU1 Serves as a Molecular Gatekeeper to Prevent In Vivo Mitochondrial Calcium OverloadJulia C Liu, Jie Liu, Kira M Holmström, et al.Pflugers Archiv : European Journal of Physiology|February 19, 2013
Sex differences in mechanisms of cardiac excitation-contraction couplingRandi J Parks, Susan E HowlettCirculation Research|May 23, 2015
The ins and outs of mitochondrial calciumToren Finkel, Sara Menazza, Kira M Holmström, et al.European Journal of Pharmacology|July 17, 2012
H-89 decreases the gain of excitation-contraction coupling and attenuates calcium sparks in the absence of beta-adrenergic stimulationRandi J Parks, Susan E HowlettCardiovascular Research|August 31, 2018
Cyclophilin D-mediated regulation of the permeability transition pore is altered in mice lacking the mitochondrial calcium uniporterRandi J Parks, Sara Menazza, Kira M Holmström, et al.Pageof 32