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Raphael S Haider

Showing results (1-10 of 9) with videos related to

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Frontiers in Cell and Developmental Biology|June 10, 2021
Differential Regulation of GPCRs-Are GRK Expression Levels the Key?Edda S F Matthees, Raphael S Haider, Carsten Hoffmann, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|April 10, 2024
S1PR3 agonism and S1P lyase inhibition rescue mice in the severe state of experimental sepsisAnke C Ziegler, Raphael S Haider, Carsten Hoffmann, et al.
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|June 1, 2023
Conformational flexibility of β-arrestins - How these scaffolding proteins guide and transform the functionality of GPCRsRaphael S Haider, Mona Reichel, Edda S F Matthees, et al.
Nature Communications|September 26, 2022
β-arrestin1 and 2 exhibit distinct phosphorylation-dependent conformations when coupling to the same GPCR in living cellsRaphael S Haider, Edda S F Matthees, Julia Drube, et al.
Scientific Reports|January 26, 2019
Arrestin-1 engineering facilitates complex stabilization with native rhodopsinRaphael S Haider, Florian Wilhelm, Aurélien Rizk, et al.
Nature Communications|July 2, 2025
Helix-bundle and C-terminal GPCR domains differentially influence GRK-specific functions and β-arrestin-mediated regulationEdda S F Matthees, Raphael S Haider, Laura Klement, et al.
Cellular and Molecular Gastroenterology and Hepatology|February 5, 2021
Circulating Bile Acids in Liver Failure Activate TGR5 and Induce Monocyte DysfunctionJulia Leonhardt, Raphael S Haider, Christoph Sponholz, et al.
Hepatology Communications|March 22, 2024
Microbially conjugated bile salts found in human bile activate the bile salt receptors TGR5 and FXRÜmran Ay, Martin Leníček, Raphael S Haider, et al.
Biomolecules|October 28, 2023
G Protein-Coupled Receptor Kinase 2 Selectively Enhances β-Arrestin Recruitment to the D<sub>2</sub> Dopamine Receptor through Mechanisms That Are Independent of Receptor PhosphorylationMarta Sánchez-Soto, Noelia M Boldizsar, Kayla A Schardien, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Frontiers in Cell and Developmental Biology|June 10, 2021
Differential Regulation of GPCRs-Are GRK Expression Levels the Key?Edda S F Matthees, Raphael S Haider, Carsten Hoffmann, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|April 10, 2024
S1PR3 agonism and S1P lyase inhibition rescue mice in the severe state of experimental sepsisAnke C Ziegler, Raphael S Haider, Carsten Hoffmann, et al.
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|June 1, 2023
Conformational flexibility of β-arrestins - How these scaffolding proteins guide and transform the functionality of GPCRsRaphael S Haider, Mona Reichel, Edda S F Matthees, et al.
Nature Communications|September 26, 2022
β-arrestin1 and 2 exhibit distinct phosphorylation-dependent conformations when coupling to the same GPCR in living cellsRaphael S Haider, Edda S F Matthees, Julia Drube, et al.
Scientific Reports|January 26, 2019
Arrestin-1 engineering facilitates complex stabilization with native rhodopsinRaphael S Haider, Florian Wilhelm, Aurélien Rizk, et al.
Nature Communications|July 2, 2025
Helix-bundle and C-terminal GPCR domains differentially influence GRK-specific functions and β-arrestin-mediated regulationEdda S F Matthees, Raphael S Haider, Laura Klement, et al.
Cellular and Molecular Gastroenterology and Hepatology|February 5, 2021
Circulating Bile Acids in Liver Failure Activate TGR5 and Induce Monocyte DysfunctionJulia Leonhardt, Raphael S Haider, Christoph Sponholz, et al.
Hepatology Communications|March 22, 2024
Microbially conjugated bile salts found in human bile activate the bile salt receptors TGR5 and FXRÜmran Ay, Martin Leníček, Raphael S Haider, et al.
Biomolecules|October 28, 2023
G Protein-Coupled Receptor Kinase 2 Selectively Enhances β-Arrestin Recruitment to the D<sub>2</sub> Dopamine Receptor through Mechanisms That Are Independent of Receptor PhosphorylationMarta Sánchez-Soto, Noelia M Boldizsar, Kayla A Schardien, et al.
Pageof 1