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Neurobiology of Pain (Cambridge, Mass.)
|
September 19, 2019
Voluntary exercise reduces both chemotherapy-induced neuropathic nociception and deficits in hippocampal cellular proliferation in a mouse model of paclitaxel-induced peripheral neuropathy
Richard A Slivicki, Sonali S Mali, Andrea G Hohmann
Pharmacological Research
|
February 11, 2019
Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro
Richard A Slivicki, Zhili Xu, Sonali S Mali, et al.
Behavioural Brain Research
|
December 3, 2016
The chemotherapeutic agent paclitaxel selectively impairs learning while sparing source memory and spatial memory
Alexandra E Smith, Richard A Slivicki, Andrea G Hohmann, et al.
Plos One
|
January 26, 2016
Impact of Genetic Reduction of NMNAT2 on Chemotherapy-Induced Losses in Cell Viability In Vitro and Peripheral Neuropathy In Vivo
Richard A Slivicki, Yousuf O Ali, Hui-Chen Lu, et al.
Pain
|
December 28, 2021
The cannabinoid agonist CB-13 produces peripherally mediated analgesia in mice but elicits tolerance and signs of central nervous system activity with repeated dosing
Richard A Slivicki, Jiwon Yi, Victoria E Brings, et al.
European Journal of Pharmacology
|
September 8, 2020
The cannabinoid CB<sub>2</sub> receptor agonist LY2828360 synergizes with morphine to suppress neuropathic nociception and attenuates morphine reward and physical dependence
Vishakh Iyer, Richard A Slivicki, Ana C Thomaz, et al.
The Journal of Pharmacology and Experimental Therapeutics
|
October 3, 2018
Brain-Permeant and -Impermeant Inhibitors of Fatty Acid Amide Hydrolase Synergize with the Opioid Analgesic Morphine to Suppress Chemotherapy-Induced Neuropathic Nociception Without Enhancing Effects of Morphine on Gastrointestinal Transit
Richard A Slivicki, Shahin A Saberi, Vishakh Iyer, et al.
Molecular Pain
|
May 15, 2016
A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolase
Lawrence M Carey, Richard A Slivicki, Emma Leishman, et al.
Frontiers in Molecular Neuroscience
|
May 16, 2020
Positive Allosteric Modulation of CB<sub>1</sub> Cannabinoid Receptor Signaling Enhances Morphine Antinociception and Attenuates Morphine Tolerance Without Enhancing Morphine- Induced Dependence or Reward
Richard A Slivicki, Vishakh Iyer, Sonali S Mali, et al.
Biorxiv : the Preprint Server for Biology
|
December 18, 2023
Impact of Δ<sup>9</sup>-Tetrahydrocannabinol and oxycodone co-administration on measures of antinociception, dependence, circadian activity, and reward in mice
Richard A Slivicki, Justin G Wang, Vy Trinh Tran Nhat, et al.
Page
of 3
Search research articles
Search
Showing results (1-10 of 25) with videos related to
Sort By:
Page
of 3
Neurobiology of Pain (Cambridge, Mass.)
|
September 19, 2019
Voluntary exercise reduces both chemotherapy-induced neuropathic nociception and deficits in hippocampal cellular proliferation in a mouse model of paclitaxel-induced peripheral neuropathy
Richard A Slivicki, Sonali S Mali, Andrea G Hohmann
Pharmacological Research
|
February 11, 2019
Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro
Richard A Slivicki, Zhili Xu, Sonali S Mali, et al.
Behavioural Brain Research
|
December 3, 2016
The chemotherapeutic agent paclitaxel selectively impairs learning while sparing source memory and spatial memory
Alexandra E Smith, Richard A Slivicki, Andrea G Hohmann, et al.
Plos One
|
January 26, 2016
Impact of Genetic Reduction of NMNAT2 on Chemotherapy-Induced Losses in Cell Viability In Vitro and Peripheral Neuropathy In Vivo
Richard A Slivicki, Yousuf O Ali, Hui-Chen Lu, et al.
Pain
|
December 28, 2021
The cannabinoid agonist CB-13 produces peripherally mediated analgesia in mice but elicits tolerance and signs of central nervous system activity with repeated dosing
Richard A Slivicki, Jiwon Yi, Victoria E Brings, et al.
European Journal of Pharmacology
|
September 8, 2020
The cannabinoid CB<sub>2</sub> receptor agonist LY2828360 synergizes with morphine to suppress neuropathic nociception and attenuates morphine reward and physical dependence
Vishakh Iyer, Richard A Slivicki, Ana C Thomaz, et al.
The Journal of Pharmacology and Experimental Therapeutics
|
October 3, 2018
Brain-Permeant and -Impermeant Inhibitors of Fatty Acid Amide Hydrolase Synergize with the Opioid Analgesic Morphine to Suppress Chemotherapy-Induced Neuropathic Nociception Without Enhancing Effects of Morphine on Gastrointestinal Transit
Richard A Slivicki, Shahin A Saberi, Vishakh Iyer, et al.
Molecular Pain
|
May 15, 2016
A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolase
Lawrence M Carey, Richard A Slivicki, Emma Leishman, et al.
Frontiers in Molecular Neuroscience
|
May 16, 2020
Positive Allosteric Modulation of CB<sub>1</sub> Cannabinoid Receptor Signaling Enhances Morphine Antinociception and Attenuates Morphine Tolerance Without Enhancing Morphine- Induced Dependence or Reward
Richard A Slivicki, Vishakh Iyer, Sonali S Mali, et al.
Biorxiv : the Preprint Server for Biology
|
December 18, 2023
Impact of Δ<sup>9</sup>-Tetrahydrocannabinol and oxycodone co-administration on measures of antinociception, dependence, circadian activity, and reward in mice
Richard A Slivicki, Justin G Wang, Vy Trinh Tran Nhat, et al.
Page
of 3