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Trends in Pharmacological Sciences|June 1, 2017
Targeting PAR1: Now What?Robert Flaumenhaft, Karen De CeunynckBlood|January 15, 2015
Parmodulins inhibit thrombus formation without inducing endothelial injury caused by vorapaxarOmozuanvbo Aisiku, Christian G Peters, Karen De Ceunynck, et al.Blood|October 25, 2012
Unwinding the von Willebrand factor strings puzzleKaren De Ceunynck, Simon F De Meyer, Karen VanhoorelbekeCurrent Opinion in Hematology|June 10, 2017
Advances in vascular thiol isomerase functionRobert FlaumenhaftProceedings of the National Academy of Sciences of the United States of America|January 19, 2018
PAR1 agonists stimulate APC-like endothelial cytoprotection and confer resistance to thromboinflammatory injuryKaren De Ceunynck, Christian G Peters, Abhishek Jain, et al.Trends in Cardiovascular Medicine|April 2, 2013
Protein disulfide isomerase as an antithrombotic targetRobert FlaumenhaftBlood Cells, Molecules & Diseases|February 10, 2006
Formation and fate of platelet microparticlesRobert FlaumenhaftArteriosclerosis, Thrombosis, and Vascular Biology|May 10, 2003
Molecular basis of platelet granule secretionRobert FlaumenhaftBlood|July 30, 2021
The secreted tyrosine kinase VLK is essential for normal platelet activation and thrombus formationLeila Revollo, Glenn Merrill-Skoloff, Karen De Ceunynck, et al.Pageof 12