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Robert Sabat

Showing results (61-70 of 129) with videos related to

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Immunotherapy|April 6, 2021
Immunotherapy in psoriasisLilla Landeck, Robert Sabat, Kamran Ghoreschi, et al.
JAMA Dermatology|February 22, 2018
Association of Hidradenitis Suppurativa With Body ImageSylke Schneider-Burrus, Agata Jost, Eva Milena Johanna Peters, et al.
Current Gene Therapy|June 19, 2009
Interleukin-10 and articular cartilage: experimental therapeutical approaches in cartilage disordersGundula Schulze-Tanzil, Hala Zreiqat, Robert Sabat, et al.
Frontiers in Medicine|May 3, 2024
Patient-reported assessment of medical care for chronic inflammatory skin diseases: an enterprise-based surveyKerstin Wolk, Maximilian Schielein, Julia-Tatjana Maul, et al.
International Immunology|March 20, 2009
Differential IL-23 requirement for IL-22 and IL-17A production during innate immunity against Salmonella enterica serovar EnteritidisSabine Siegemund, Nicole Schütze, Silke Schulz, et al.
Journal of Personalized Medicine|November 27, 2021
The Effect of TNF-α Inhibitors on Nail Psoriasis and Psoriatic Arthritis-Real-World Data from Dermatology PracticeGeorgios Kokolakis, Robert Sabat, Imma Fischer, et al.
Frontiers in Medicine|May 14, 2021
Features Associated With Quality of Life Impairment in Hidradenitis Suppurativa PatientsSylke Schneider-Burrus, Athanasia Tsaousi, Sebastian Barbus, et al.
Journal of Leukocyte Biology|April 24, 2009
IL-10 protects monocytes and macrophages from complement-mediated lysisNadine Koch, Mechthild Jung, Robert Sabat, et al.
Experimental Dermatology|April 19, 2003
Keratinocyte unresponsiveness towards interleukin-10: lack of specific binding due to deficient IL-10 receptor 1 expressionMartina Seifert, Beate H Gruenberg, Robert Sabat, et al.
The Journal of Investigative Dermatology|November 25, 2003
Ultraviolet B radiation-mediated inhibition of interferon-gamma-induced keratinocyte activation is independent of interleukin-10 and other soluble mediators but associated with enhanced intracellular suppressors of cytokine-signaling expressionMarkus Friedrich, Ruth Holzmann, Wolfram Sterry, et al.
Pageof 13

Showing results (61-70 of 129) with videos related to

Sort By:
Pageof 13
Immunotherapy|April 6, 2021
Immunotherapy in psoriasisLilla Landeck, Robert Sabat, Kamran Ghoreschi, et al.
JAMA Dermatology|February 22, 2018
Association of Hidradenitis Suppurativa With Body ImageSylke Schneider-Burrus, Agata Jost, Eva Milena Johanna Peters, et al.
Current Gene Therapy|June 19, 2009
Interleukin-10 and articular cartilage: experimental therapeutical approaches in cartilage disordersGundula Schulze-Tanzil, Hala Zreiqat, Robert Sabat, et al.
Frontiers in Medicine|May 3, 2024
Patient-reported assessment of medical care for chronic inflammatory skin diseases: an enterprise-based surveyKerstin Wolk, Maximilian Schielein, Julia-Tatjana Maul, et al.
International Immunology|March 20, 2009
Differential IL-23 requirement for IL-22 and IL-17A production during innate immunity against Salmonella enterica serovar EnteritidisSabine Siegemund, Nicole Schütze, Silke Schulz, et al.
Journal of Personalized Medicine|November 27, 2021
The Effect of TNF-α Inhibitors on Nail Psoriasis and Psoriatic Arthritis-Real-World Data from Dermatology PracticeGeorgios Kokolakis, Robert Sabat, Imma Fischer, et al.
Frontiers in Medicine|May 14, 2021
Features Associated With Quality of Life Impairment in Hidradenitis Suppurativa PatientsSylke Schneider-Burrus, Athanasia Tsaousi, Sebastian Barbus, et al.
Journal of Leukocyte Biology|April 24, 2009
IL-10 protects monocytes and macrophages from complement-mediated lysisNadine Koch, Mechthild Jung, Robert Sabat, et al.
Experimental Dermatology|April 19, 2003
Keratinocyte unresponsiveness towards interleukin-10: lack of specific binding due to deficient IL-10 receptor 1 expressionMartina Seifert, Beate H Gruenberg, Robert Sabat, et al.
The Journal of Investigative Dermatology|November 25, 2003
Ultraviolet B radiation-mediated inhibition of interferon-gamma-induced keratinocyte activation is independent of interleukin-10 and other soluble mediators but associated with enhanced intracellular suppressors of cytokine-signaling expressionMarkus Friedrich, Ruth Holzmann, Wolfram Sterry, et al.
Pageof 13