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S B Bratton

Showing results (1-10 of 11) with videos related to

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Trends in Pharmacological Sciences|June 8, 2001
Apoptotic death sensor: an organelle's alter ego?S B Bratton, G M Cohen
Cellular and Molecular Life Sciences : CMLS|June 19, 2007
'Heated' debates in apoptosisR S Milleron, S B Bratton
Cell Death and Differentiation|February 20, 2010
TRAIL-activated stress kinases suppress apoptosis through transcriptional upregulation of MCL-1J K Son, S Varadarajan, S B Bratton
Chemical Research in Toxicology|July 18, 2000
The putative benzene metabolite 2,3, 5-tris(glutathion-S-yl)hydroquinone depletes glutathione, stimulates sphingomyelin turnover, and induces apoptosis in HL-60 cellsS B Bratton, S S Lau, T J Monks
Chemical Research in Toxicology|August 1, 1997
Identification of quinol thioethers in bone marrow of hydroquinone/phenol-treated rats and mice and their potential role in benzene-mediated hematotoxicityS B Bratton, S S Lau, T J Monks
Experimental Cell Research|March 31, 2000
Protein complexes activate distinct caspase cascades in death receptor and stress-induced apoptosisS B Bratton, M MacFarlane, K Cain, et al.
Cell Death and Differentiation|September 11, 2001
Caspase-3 cleaves Apaf-1 into an approximately 30 kDa fragment that associates with an inappropriately oligomerized and biologically inactive approximately 1.4 MDa apoptosome complexS B Bratton, G Walker, D L Roberts, et al.
Cell Death and Differentiation|August 16, 2002
XIAP inhibition of caspase-3 preserves its association with the Apaf-1 apoptosome and prevents CD95- and Bax-induced apoptosisS B Bratton, J Lewis, M Butterworth, et al.
The Journal of Biological Chemistry|February 29, 2000
Apaf-1 oligomerizes into biologically active approximately 700-kDa and inactive approximately 1.4-MDa apoptosome complexesK Cain, S B Bratton, C Langlais, et al.
The EMBO Journal|March 7, 2001
Recruitment, activation and retention of caspases-9 and -3 by Apaf-1 apoptosome and associated XIAP complexesS B Bratton, G Walker, S M Srinivasula, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Trends in Pharmacological Sciences|June 8, 2001
Apoptotic death sensor: an organelle's alter ego?S B Bratton, G M Cohen
Cellular and Molecular Life Sciences : CMLS|June 19, 2007
'Heated' debates in apoptosisR S Milleron, S B Bratton
Cell Death and Differentiation|February 20, 2010
TRAIL-activated stress kinases suppress apoptosis through transcriptional upregulation of MCL-1J K Son, S Varadarajan, S B Bratton
Chemical Research in Toxicology|July 18, 2000
The putative benzene metabolite 2,3, 5-tris(glutathion-S-yl)hydroquinone depletes glutathione, stimulates sphingomyelin turnover, and induces apoptosis in HL-60 cellsS B Bratton, S S Lau, T J Monks
Chemical Research in Toxicology|August 1, 1997
Identification of quinol thioethers in bone marrow of hydroquinone/phenol-treated rats and mice and their potential role in benzene-mediated hematotoxicityS B Bratton, S S Lau, T J Monks
Experimental Cell Research|March 31, 2000
Protein complexes activate distinct caspase cascades in death receptor and stress-induced apoptosisS B Bratton, M MacFarlane, K Cain, et al.
Cell Death and Differentiation|September 11, 2001
Caspase-3 cleaves Apaf-1 into an approximately 30 kDa fragment that associates with an inappropriately oligomerized and biologically inactive approximately 1.4 MDa apoptosome complexS B Bratton, G Walker, D L Roberts, et al.
Cell Death and Differentiation|August 16, 2002
XIAP inhibition of caspase-3 preserves its association with the Apaf-1 apoptosome and prevents CD95- and Bax-induced apoptosisS B Bratton, J Lewis, M Butterworth, et al.
The Journal of Biological Chemistry|February 29, 2000
Apaf-1 oligomerizes into biologically active approximately 700-kDa and inactive approximately 1.4-MDa apoptosome complexesK Cain, S B Bratton, C Langlais, et al.
The EMBO Journal|March 7, 2001
Recruitment, activation and retention of caspases-9 and -3 by Apaf-1 apoptosome and associated XIAP complexesS B Bratton, G Walker, S M Srinivasula, et al.
Pageof 2