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Science (New York, N.Y.)|October 23, 1987
A cytoplasmic protein stimulates normal N-ras p21 GTPase, but does not affect oncogenic mutantsM Trahey, F McCormickThe EMBO Journal|October 2, 1995
Bcr and Raf form a complex in vivo via 14-3-3 proteinsS Braselmann, F McCormickCancer Surveys|January 1, 1992
Interactions between p21ras proteins and their GTPase activating proteinsP Polakis, F McCormickNature|June 13, 1991
Differential regulation of rasGAP and neurofibromatosis gene product activitiesG Bollag, F McCormickMolecular Medicine Today|December 1, 1996
Replicating viruses as selective cancer therapeuticsD H Kirn, F McCormickArchives of Neurology|May 1, 1978
Intracerebral venous angioma. Case report and reviewM Sarwar, W F McCormickScience (New York, N.Y.)|November 12, 1993
Inhibition by cAMP of Ras-dependent activation of RafS J Cook, F McCormickThe Biochemical Journal|November 15, 1996
Kinetic and biochemical correlation between sustained p44ERK1 (44 kDa extracellular signal-regulated kinase 1) activation and lysophosphatidic acid-stimulated DNA synthesis in Rat-1 cellsS J Cook, F McCormickCancer Research|June 5, 2001
Selective targeting to the hyperactive beta-catenin/T-cell factor pathway in colon cancer cellsR H Chen, F McCormickPageof 132