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The Journal of Biological Chemistry
|
February 10, 1973
Hemoglobin 3 chains in apes. Primary structures and the presumptive nature of back mutation in a normally silent gene
S H Boyer, A N Noyes, M L Boyer, et al.
Cell
|
July 1, 1979
SV40 recombinants carrying rabbit beta-globin gene coding sequences
D H Hamer, K D Smith, S H Boyer, et al.
Blood
|
May 1, 1989
Erythropoietin receptors on murine erythroid colony-forming units: natural history
K T Landschulz, A N Noyes, O Rogers, et al.
Canadian Journal of Genetics and Cytology. Journal Canadien De Genetique Et De Cytologie
|
March 1, 1978
The proportion of all point mutations which are unacceptable: an estimate based on hemoglobin amino acid and nucleotide sequences
S H Boyer, A F Scott, L M Kunkel, et al.
Nucleic Acids Research
|
February 25, 1989
Cloning and sequence of mouse erythroid delta-aminolevulinate dehydratase cDNA
T R Bishop, Z I Hodes, L P Frelin, et al.
Blood
|
September 1, 1985
5-Azacytidine increases HbF production and reduces anemia in sickle cell disease: dose-response analysis of subcutaneous and oral dosage regimens
G J Dover, S Charache, S H Boyer, et al.
The Journal of Biological Chemistry
|
February 25, 1983
Adjuvants to immunological methods for mRNA purification. Application to the isolation of mRNA for carbonic anhydrase I from rabbit reticulocytes
S H Boyer, K D Smith, A N Noyes, et al.
The Journal of Biological Chemistry
|
November 25, 1974
Immunological characterization of rabbit hemoglobin alpha and beta chain-synthesizing polysomes
S H Boyer, K D Smith, A N Noyes, et al.
Annals of the New York Academy of Sciences
|
January 1, 1984
Isolation of cDNA clones for rabbit red cell carbonic anhydrase and catalase: a pilot study directed at isolation of coordinately expressed genes
S H Boyer, H Ostrer, K D Smith, et al.
Progress in Clinical and Biological Research
|
January 1, 1983
5-Azacytidine increases fetal hemoglobin production in a patient with sickle cell disease
G J Dover, S H Charache, S H Boyer, et al.
Page
of 6
Search research articles
Search
Showing results (31-40 of 58) with videos related to
Sort By:
Page
of 6
The Journal of Biological Chemistry
|
February 10, 1973
Hemoglobin 3 chains in apes. Primary structures and the presumptive nature of back mutation in a normally silent gene
S H Boyer, A N Noyes, M L Boyer, et al.
Cell
|
July 1, 1979
SV40 recombinants carrying rabbit beta-globin gene coding sequences
D H Hamer, K D Smith, S H Boyer, et al.
Blood
|
May 1, 1989
Erythropoietin receptors on murine erythroid colony-forming units: natural history
K T Landschulz, A N Noyes, O Rogers, et al.
Canadian Journal of Genetics and Cytology. Journal Canadien De Genetique Et De Cytologie
|
March 1, 1978
The proportion of all point mutations which are unacceptable: an estimate based on hemoglobin amino acid and nucleotide sequences
S H Boyer, A F Scott, L M Kunkel, et al.
Nucleic Acids Research
|
February 25, 1989
Cloning and sequence of mouse erythroid delta-aminolevulinate dehydratase cDNA
T R Bishop, Z I Hodes, L P Frelin, et al.
Blood
|
September 1, 1985
5-Azacytidine increases HbF production and reduces anemia in sickle cell disease: dose-response analysis of subcutaneous and oral dosage regimens
G J Dover, S Charache, S H Boyer, et al.
The Journal of Biological Chemistry
|
February 25, 1983
Adjuvants to immunological methods for mRNA purification. Application to the isolation of mRNA for carbonic anhydrase I from rabbit reticulocytes
S H Boyer, K D Smith, A N Noyes, et al.
The Journal of Biological Chemistry
|
November 25, 1974
Immunological characterization of rabbit hemoglobin alpha and beta chain-synthesizing polysomes
S H Boyer, K D Smith, A N Noyes, et al.
Annals of the New York Academy of Sciences
|
January 1, 1984
Isolation of cDNA clones for rabbit red cell carbonic anhydrase and catalase: a pilot study directed at isolation of coordinately expressed genes
S H Boyer, H Ostrer, K D Smith, et al.
Progress in Clinical and Biological Research
|
January 1, 1983
5-Azacytidine increases fetal hemoglobin production in a patient with sickle cell disease
G J Dover, S H Charache, S H Boyer, et al.
Page
of 6