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Archives of Virology. Supplementum
|
January 1, 1994
Coronavirus polyprotein processing
S R Weiss, S A Hughes, P J Bonilla, et al.
Microbial Pathogenesis
|
August 1, 1987
Coronavirus mouse hepatitis virus (MHV)-A59 causes a persistent, productive infection in primary glial cell cultures
E Lavi, A Suzumura, M Hirayama, et al.
Brain Research
|
June 1, 1988
mRNA levels of all three neurofilament proteins decline following nerve transection
M E Goldstein, S R Weiss, R A Lazzarini, et al.
Brain Research
|
May 7, 1986
CRF-induced seizures and behavior: interaction with amygdala kindling
S R Weiss, R M Post, P W Gold, et al.
Nucleic Acids Research
|
April 11, 1990
The primary structure and expression of the second open reading frame of the polymerase gene of the coronavirus MHV-A59; a highly conserved polymerase is expressed by an efficient ribosomal frameshifting mechanism
P J Bredenbeek, C J Pachuk, A F Noten, et al.
Journal of Neurovirology
|
March 5, 1999
Localization of mouse hepatitis virus open reading frame 1A derived proteins
W Bi, J D Piñón, S Hughes, et al.
Advances in Experimental Medicine and Biology
|
January 5, 2002
Role of the spike protein in murine coronavirus induced hepatitis: an in vivo study using targeted RNA recombination
S Navas, S H Seo, M M Chua, et al.
Epilepsia
|
September 1, 1985
Chronic carbamazepine treatment increases brain adenosine receptors
P J Marangos, S R Weiss, P Montgomery, et al.
Advances in Experimental Medicine and Biology
|
January 1, 1987
Expression of MHC class I genes in mouse hepatitis virus (MHV-A59) infection and in multiple sclerosis
E Lavi, A Suzumura, L A Lampson, et al.
Brain Research
|
January 16, 1997
Amygdala-kindled seizures increase the expression of corticotropin-releasing factor (CRF) and CRF-binding protein in GABAergic interneurons of the dentate hilus
M A Smith, S R Weiss, R L Berry, et al.
Page
of 22
Search research articles
Search
Showing results (181-190 of 216) with videos related to
Sort By:
Page
of 22
Archives of Virology. Supplementum
|
January 1, 1994
Coronavirus polyprotein processing
S R Weiss, S A Hughes, P J Bonilla, et al.
Microbial Pathogenesis
|
August 1, 1987
Coronavirus mouse hepatitis virus (MHV)-A59 causes a persistent, productive infection in primary glial cell cultures
E Lavi, A Suzumura, M Hirayama, et al.
Brain Research
|
June 1, 1988
mRNA levels of all three neurofilament proteins decline following nerve transection
M E Goldstein, S R Weiss, R A Lazzarini, et al.
Brain Research
|
May 7, 1986
CRF-induced seizures and behavior: interaction with amygdala kindling
S R Weiss, R M Post, P W Gold, et al.
Nucleic Acids Research
|
April 11, 1990
The primary structure and expression of the second open reading frame of the polymerase gene of the coronavirus MHV-A59; a highly conserved polymerase is expressed by an efficient ribosomal frameshifting mechanism
P J Bredenbeek, C J Pachuk, A F Noten, et al.
Journal of Neurovirology
|
March 5, 1999
Localization of mouse hepatitis virus open reading frame 1A derived proteins
W Bi, J D Piñón, S Hughes, et al.
Advances in Experimental Medicine and Biology
|
January 5, 2002
Role of the spike protein in murine coronavirus induced hepatitis: an in vivo study using targeted RNA recombination
S Navas, S H Seo, M M Chua, et al.
Epilepsia
|
September 1, 1985
Chronic carbamazepine treatment increases brain adenosine receptors
P J Marangos, S R Weiss, P Montgomery, et al.
Advances in Experimental Medicine and Biology
|
January 1, 1987
Expression of MHC class I genes in mouse hepatitis virus (MHV-A59) infection and in multiple sclerosis
E Lavi, A Suzumura, L A Lampson, et al.
Brain Research
|
January 16, 1997
Amygdala-kindled seizures increase the expression of corticotropin-releasing factor (CRF) and CRF-binding protein in GABAergic interneurons of the dentate hilus
M A Smith, S R Weiss, R L Berry, et al.
Page
of 22