Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

S Schnittger

Showing results (41-50 of 131) with videos related to

Pageof 14
Sort By:
Leukemia|September 5, 2012
A rare but specific subset of adult AML patients can be defined by the cytogenetically cryptic NUP98-NSD1 fusion geneA Fasan, C Haferlach, T Alpermann, et al.
Genomics|November 1, 1992
Pax1, a member of the paired box-containing class of developmental control genes, is mapped to human chromosome 20p11.2 by in situ hybridization (ISH and FISH)S Schnittger, V V Rao, U Deutsch, et al.
Human Genetics|June 1, 1991
A sterile male with 45,X0 and a Y;22 translocationJ Arnemann, S Schnittger, G K Hinkel, et al.
Human Genetics|May 1, 1994
The human vigilin gene: identification, chromosomal localization and expression patternG Plenz, S Kügler, S Schnittger, et al.
Leukemia|October 3, 2002
Karyotype instability between diagnosis and relapse in 117 patients with acute myeloid leukemia: implications for resistance against therapyW Kern, T Haferlach, S Schnittger, et al.
Bone Marrow Transplantation|July 1, 2008
Minimal residual disease diagnostics in myeloid malignancies in the post transplant periodU Bacher, A R Zander, T Haferlach, et al.
Der Pathologe|October 23, 2012
[Diagnostics of acute leukemias: interaction of phenotypic and genetic methods]U Bacher, C Haferlach, S Schnittger, et al.
Leukemia|May 20, 2005
Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 8, 11, or 13, monosomy 7, or deletion 5qC Schoch, A Kohlmann, M Dugas, et al.
Leukemia|April 9, 2005
New insights into MLL gene rearranged acute leukemias using gene expression profiling: shared pathways, lineage commitment, and partner genesA Kohlmann, C Schoch, M Dugas, et al.
Leukemia|October 22, 2011
Deletion of the tumor-suppressor gene NF1 occurs in 5% of myeloid malignancies and is accompanied by a mutation in the remaining allele in half of the casesC Haferlach, V Grossmann, A Kohlmann, et al.
Pageof 14

Showing results (41-50 of 131) with videos related to

Sort By:
Pageof 14
Leukemia|September 5, 2012
A rare but specific subset of adult AML patients can be defined by the cytogenetically cryptic NUP98-NSD1 fusion geneA Fasan, C Haferlach, T Alpermann, et al.
Genomics|November 1, 1992
Pax1, a member of the paired box-containing class of developmental control genes, is mapped to human chromosome 20p11.2 by in situ hybridization (ISH and FISH)S Schnittger, V V Rao, U Deutsch, et al.
Human Genetics|June 1, 1991
A sterile male with 45,X0 and a Y;22 translocationJ Arnemann, S Schnittger, G K Hinkel, et al.
Human Genetics|May 1, 1994
The human vigilin gene: identification, chromosomal localization and expression patternG Plenz, S Kügler, S Schnittger, et al.
Leukemia|October 3, 2002
Karyotype instability between diagnosis and relapse in 117 patients with acute myeloid leukemia: implications for resistance against therapyW Kern, T Haferlach, S Schnittger, et al.
Bone Marrow Transplantation|July 1, 2008
Minimal residual disease diagnostics in myeloid malignancies in the post transplant periodU Bacher, A R Zander, T Haferlach, et al.
Der Pathologe|October 23, 2012
[Diagnostics of acute leukemias: interaction of phenotypic and genetic methods]U Bacher, C Haferlach, S Schnittger, et al.
Leukemia|May 20, 2005
Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 8, 11, or 13, monosomy 7, or deletion 5qC Schoch, A Kohlmann, M Dugas, et al.
Leukemia|April 9, 2005
New insights into MLL gene rearranged acute leukemias using gene expression profiling: shared pathways, lineage commitment, and partner genesA Kohlmann, C Schoch, M Dugas, et al.
Leukemia|October 22, 2011
Deletion of the tumor-suppressor gene NF1 occurs in 5% of myeloid malignancies and is accompanied by a mutation in the remaining allele in half of the casesC Haferlach, V Grossmann, A Kohlmann, et al.
Pageof 14