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Plos Pathogens|January 18, 2013
Glucose phosphorylation is required for Mycobacterium tuberculosis persistence in miceJoeli Marrero, Carolina Trujillo, Kyu Y Rhee, et al.
Microbiology Resource Announcements|July 11, 2025
Identifying genetic determinants of Mycobacterium tuberculosis acid growth arrest by transposon mutagenesis coupled with next-generation sequencing (Tn-seq)Claire Healy, Thomas R Ioerger, Sabine Ehrt, et al.
Cell|July 23, 2021
Genome-wide gene expression tuning reveals diverse vulnerabilities of M. tuberculosisBarbara Bosch, Michael A DeJesus, Nicholas C Poulton, et al.
Nature Communications|March 22, 2026
Activation of L-histidine biosynthesis as a new antibiotic strategy against Mycobacterium tuberculosisDebbie M Hunt, João Pedro Pisco, Angela Rodgers, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 6, 2013
A genetic strategy to identify targets for the development of drugs that prevent bacterial persistenceJee-Hyun Kim, Kathryn M O'Brien, Ritu Sharma, et al.
Nucleic Acids Research|June 12, 2003
Single-chain Tet transregulatorsChristel Krueger, Christian Berens, Andreas Schmidt, et al.
Annals of the New York Academy of Sciences|April 10, 2024
Weak links: Advancing target-based drug discovery by identifying the most vulnerable targetsBarbara Bosch, Michael A DeJesus, Dirk Schnappinger, et al.
Mbio|January 26, 2017
PPE Surface Proteins Are Required for Heme Utilization by Mycobacterium tuberculosisAvishek Mitra, Alexander Speer, Kan Lin, et al.
Tuberculosis (Edinburgh, Scotland)|July 23, 2011
Mycobacterium tuberculosis gene Rv2136c is dispensable for acid resistance and virulence in miceCrystal M Darby, Aditya Venugopal, Sabine Ehrt, et al.
Immunology|October 21, 2005
Induction of macrophage-derived SLPI by Mycobacterium tuberculosis depends on TLR2 but not MyD88Aihao Ding, Hongwei Yu, Jingxuan Yang, et al.
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