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Mbio|January 26, 2017
PPE Surface Proteins Are Required for Heme Utilization by Mycobacterium tuberculosisAvishek Mitra, Alexander Speer, Kan Lin, et al.
Tuberculosis (Edinburgh, Scotland)|July 23, 2011
Mycobacterium tuberculosis gene Rv2136c is dispensable for acid resistance and virulence in miceCrystal M Darby, Aditya Venugopal, Sabine Ehrt, et al.
Nature Medicine|December 7, 2007
In vivo gene silencing identifies the Mycobacterium tuberculosis proteasome as essential for the bacteria to persist in miceSheetal Gandotra, Dirk Schnappinger, Mercedes Monteleone, et al.
Immunology|October 21, 2005
Induction of macrophage-derived SLPI by Mycobacterium tuberculosis depends on TLR2 but not MyD88Aihao Ding, Hongwei Yu, Jingxuan Yang, et al.
Infection and Immunity|August 22, 2007
Nucleotide-binding oligomerization domain protein 2-deficient mice control infection with Mycobacterium tuberculosisSheetal Gandotra, Sihyug Jang, Peter J Murray, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 5, 2010
Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infectionJoeli Marrero, Kyu Y Rhee, Dirk Schnappinger, et al.
Elife|November 22, 2019
Persistent Mycobacterium tuberculosis infection in mice requires PerM for successful cell divisionRuojun Wang, Kaj Kreutzfeldt, Helene Botella, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 3, 2021
Growth of Mycobacterium tuberculosis at acidic pH depends on lipid assimilation and is accompanied by reduced GAPDH activityAlexandre Gouzy, Claire Healy, Katherine A Black, et al.
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