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Sandra Milasta

Showing results (11-20 of 22) with videos related to

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Blood|December 7, 2011
Mitochondrial dysfunction in ataxia-telangiectasiaYasmine A Valentin-Vega, Kirsteen H Maclean, Jacqueline Tait-Mulder, et al.
Cancer & Metabolism|February 22, 2023
Breast cancer cells that preferentially metastasize to lung or bone are more glycolytic, synthesize serine at greater rates, and consume less ATP and NADPH than parent MDA-MB-231 cellsMika B Jekabsons, Mollie Merrell, Anna G Skubiz, et al.
Cell Reports|October 8, 2013
Protective roles for caspase-8 and cFLIP in adult homeostasisRicardo Weinlich, Andrew Oberst, Christopher P Dillon, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 12, 2014
Metabolic reprogramming is required for antibody production that is suppressed in anergic but exaggerated in chronically BAFF-exposed B cellsAlfredo Caro-Maldonado, Ruoning Wang, Amanda G Nichols, et al.
Nature Cell Biology|May 1, 2012
Anti-apoptotic MCL-1 localizes to the mitochondrial matrix and couples mitochondrial fusion to respirationRhonda M Perciavalle, Daniel P Stewart, Brian Koss, et al.
Immunity|January 23, 2016
Apoptosis-Inducing-Factor-Dependent Mitochondrial Function Is Required for T Cell but Not B Cell FunctionSandra Milasta, Christopher P Dillon, Oliver E Sturm, et al.
Cell Reports|November 26, 2013
Widespread mitochondrial depletion via mitophagy does not compromise necroptosisStephen W G Tait, Andrew Oberst, Giovanni Quarato, et al.
Blood|November 21, 2014
Mito-protective autophagy is impaired in erythroid cells of aged mtDNA-mutator miceXiuJie Li-Harms, Sandra Milasta, John Lynch, et al.
Cell Reports|May 25, 2017
Targeting Metabolic Reprogramming by Influenza Infection for Therapeutic InterventionHeather S Smallwood, Susu Duan, Marie Morfouace, et al.
Cell|June 2, 2007
GAPDH and autophagy preserve survival after apoptotic cytochrome c release in the absence of caspase activationAnna Colell, Jean-Ehrland Ricci, Stephen Tait, et al.
Pageof 3

Showing results (11-20 of 22) with videos related to

Sort By:
Pageof 3
Blood|December 7, 2011
Mitochondrial dysfunction in ataxia-telangiectasiaYasmine A Valentin-Vega, Kirsteen H Maclean, Jacqueline Tait-Mulder, et al.
Cancer & Metabolism|February 22, 2023
Breast cancer cells that preferentially metastasize to lung or bone are more glycolytic, synthesize serine at greater rates, and consume less ATP and NADPH than parent MDA-MB-231 cellsMika B Jekabsons, Mollie Merrell, Anna G Skubiz, et al.
Cell Reports|October 8, 2013
Protective roles for caspase-8 and cFLIP in adult homeostasisRicardo Weinlich, Andrew Oberst, Christopher P Dillon, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 12, 2014
Metabolic reprogramming is required for antibody production that is suppressed in anergic but exaggerated in chronically BAFF-exposed B cellsAlfredo Caro-Maldonado, Ruoning Wang, Amanda G Nichols, et al.
Nature Cell Biology|May 1, 2012
Anti-apoptotic MCL-1 localizes to the mitochondrial matrix and couples mitochondrial fusion to respirationRhonda M Perciavalle, Daniel P Stewart, Brian Koss, et al.
Immunity|January 23, 2016
Apoptosis-Inducing-Factor-Dependent Mitochondrial Function Is Required for T Cell but Not B Cell FunctionSandra Milasta, Christopher P Dillon, Oliver E Sturm, et al.
Cell Reports|November 26, 2013
Widespread mitochondrial depletion via mitophagy does not compromise necroptosisStephen W G Tait, Andrew Oberst, Giovanni Quarato, et al.
Blood|November 21, 2014
Mito-protective autophagy is impaired in erythroid cells of aged mtDNA-mutator miceXiuJie Li-Harms, Sandra Milasta, John Lynch, et al.
Cell Reports|May 25, 2017
Targeting Metabolic Reprogramming by Influenza Infection for Therapeutic InterventionHeather S Smallwood, Susu Duan, Marie Morfouace, et al.
Cell|June 2, 2007
GAPDH and autophagy preserve survival after apoptotic cytochrome c release in the absence of caspase activationAnna Colell, Jean-Ehrland Ricci, Stephen Tait, et al.
Pageof 3