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Sandra Pacios

Showing results (1-10 of 14) with videos related to

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Frontiers of Oral Biology|November 25, 2015
Bone Remodeling Under Pathological ConditionsWenmei Xiao, Shuai Li, Sandra Pacios, et al.
Frontiers of Oral Biology|November 25, 2015
Cellular and Molecular Aspects of Bone RemodelingWenmei Xiao, Yu Wang, Sandra Pacios, et al.
The Journal of Cell Biology|October 23, 2013
FOXO1 promotes wound healing through the up-regulation of TGF-β1 and prevention of oxidative stressBhaskar Ponugoti, Fanxing Xu, Chenying Zhang, et al.
World Journal of Microbiology & Biotechnology|August 2, 2023
Trichoderma as a biological control agent: mechanisms of action, benefits for crops and development of formulationsSalvador A Saldaña-Mendoza, Sandra Pacios-Michelena, Arturo S Palacios-Ponce, et al.
The American Journal of Pathology|March 22, 2015
FOXO1 deletion reduces dendritic cell function and enhances susceptibility to periodontitisWenmei Xiao, Guangyu Dong, Sandra Pacios, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 20, 2011
Diabetes aggravates periodontitis by limiting repair through enhanced inflammationSandra Pacios, Jun Kang, Johnah Galicia, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 20, 2015
FOXO1 regulates dendritic cell activity through ICAM-1 and CCR7Guangyu Dong, Yu Wang, Wenmei Xiao, et al.
Scientific Reports|December 16, 2015
Osteoblast Lineage Cells Play an Essential Role in Periodontal Bone Loss Through Activation of Nuclear Factor-Kappa BSandra Pacios, Wenmei Xiao, Marcelo Mattos, et al.
Infection and Immunity|March 28, 2012
Aggregatibacter actinomycetemcomitans infection enhances apoptosis in vivo through a caspase-3-dependent mechanism in experimental periodontitisJun Kang, Beatriz de Brito Bezerra, Sandra Pacios, et al.
The American Journal of Pathology|October 12, 2013
Bacterial infection increases periodontal bone loss in diabetic rats through enhanced apoptosisSandra Pacios, Oelisoa Andriankaja, Jun Kang, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Frontiers of Oral Biology|November 25, 2015
Bone Remodeling Under Pathological ConditionsWenmei Xiao, Shuai Li, Sandra Pacios, et al.
Frontiers of Oral Biology|November 25, 2015
Cellular and Molecular Aspects of Bone RemodelingWenmei Xiao, Yu Wang, Sandra Pacios, et al.
The Journal of Cell Biology|October 23, 2013
FOXO1 promotes wound healing through the up-regulation of TGF-β1 and prevention of oxidative stressBhaskar Ponugoti, Fanxing Xu, Chenying Zhang, et al.
World Journal of Microbiology & Biotechnology|August 2, 2023
Trichoderma as a biological control agent: mechanisms of action, benefits for crops and development of formulationsSalvador A Saldaña-Mendoza, Sandra Pacios-Michelena, Arturo S Palacios-Ponce, et al.
The American Journal of Pathology|March 22, 2015
FOXO1 deletion reduces dendritic cell function and enhances susceptibility to periodontitisWenmei Xiao, Guangyu Dong, Sandra Pacios, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 20, 2011
Diabetes aggravates periodontitis by limiting repair through enhanced inflammationSandra Pacios, Jun Kang, Johnah Galicia, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 20, 2015
FOXO1 regulates dendritic cell activity through ICAM-1 and CCR7Guangyu Dong, Yu Wang, Wenmei Xiao, et al.
Scientific Reports|December 16, 2015
Osteoblast Lineage Cells Play an Essential Role in Periodontal Bone Loss Through Activation of Nuclear Factor-Kappa BSandra Pacios, Wenmei Xiao, Marcelo Mattos, et al.
Infection and Immunity|March 28, 2012
Aggregatibacter actinomycetemcomitans infection enhances apoptosis in vivo through a caspase-3-dependent mechanism in experimental periodontitisJun Kang, Beatriz de Brito Bezerra, Sandra Pacios, et al.
The American Journal of Pathology|October 12, 2013
Bacterial infection increases periodontal bone loss in diabetic rats through enhanced apoptosisSandra Pacios, Oelisoa Andriankaja, Jun Kang, et al.
Pageof 2