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Methods in Molecular Medicine|November 7, 2006
Animal models for cardiac arrhythmiasSandro L Yong, Qing K WangBiochemical and Biophysical Research Communications|February 16, 2007
Cardiac-specific overexpression of SCN5A gene leads to shorter P wave duration and PR interval in transgenic miceTeng Zhang, Sandro L Yong, Xiao-Li Tian, et al.Biochemical and Biophysical Research Communications|November 23, 2006
Characterization of the cardiac sodium channel SCN5A mutation, N1325S, in single murine ventricular myocytesSandro L Yong, Ying Ni, Teng Zhang, et al.European Journal of Pharmacology|December 21, 2018
Molecular charge associated with antiarrhythmic actions in a series of amino-2-cyclohexyl ester derivativesMichael K Pugsley, Sandro L Yong, Alan L Goldin, et al.Biochemical and Biophysical Research Communications|December 13, 2006
Optical mapping of ventricular arrhythmias in LQTS mice with SCN5A mutation N1325SXiao-Li Tian, Yuanna Cheng, Teng Zhang, et al.International Journal of Cardiology|September 19, 2009
LQTS mutation N1325S in cardiac sodium channel gene SCN5A causes cardiomyocyte apoptosis, cardiac fibrosis and contractile dysfunction in miceTeng Zhang, Sandro L Yong, Jeanne K Drinko, et al.Circulation. Arrhythmia and Electrophysiology|February 20, 2013
MOG1 rescues defective trafficking of Na(v)1.5 mutations in Brugada syndrome and sick sinus syndromeSusmita Chakrabarti, Xiaofen Wu, Zhaogang Yang, et al.Journal of Molecular Biology|November 27, 2007
Prevention of cardiac hypertrophy and heart failure by silencing of NF-kappaBSudhiranjan Gupta, David Young, Ratan K Maitra, et al.Cardiovascular Research|January 23, 2004
Mechanisms by which SCN5A mutation N1325S causes cardiac arrhythmias and sudden death in vivoXiao-Li Tian, Sandro L Yong, Xiaoping Wan, et al.Cell|December 17, 2008
Mutation in nuclear pore component NUP155 leads to atrial fibrillation and early sudden cardiac deathXianqin Zhang, Shenghan Chen, Shin Yoo, et al.Pageof 2