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The Journal of Physiology|May 20, 2009
New roles of calsequestrin and triadin in cardiac muscleBjörn C KnollmannCirculation Research|April 10, 2013
Induced pluripotent stem cell-derived cardiomyocytes: boutique science or valuable arrhythmia model?Björn C KnollmannJournal of Cardiovascular Electrophysiology|July 1, 2009
Cardiac calsequestrin: the new kid on the block in arrhythmiasNagesh Chopra, Björn C KnollmannCurrent Opinion in Cardiology|March 25, 2011
Genetics of sudden cardiac death syndromesNagesh Chopra, Björn C KnollmannAmerican Journal of Physiology. Heart and Circulatory Physiology|December 27, 2011
Calsequestrin 2 and arrhythmiasMichela Faggioni, Björn C KnollmannThe Journal of Clinical Investigation|July 26, 2011
Oxidized CaMKII: a "heart stopper" for the sinus node?Sabine Huke, Björn C KnollmannJournal of Molecular and Cellular Cardiology|January 26, 2010
Increased myofilament Ca2+-sensitivity and arrhythmia susceptibilitySabine Huke, Björn C KnollmannJournal of Electrocardiology|August 30, 2011
Mechanism underlying catecholaminergic polymorphic ventricular tachycardia and approaches to therapyHiroshi Watanabe, Björn C KnollmannCardiovascular Research|February 12, 2013
Triadin regulates cardiac muscle couplon structure and microdomain Ca(2+) signalling: a path towards ventricular arrhythmiasNagesh Chopra, Björn C KnollmannAnnual Review of Pharmacology and Toxicology|October 7, 2024
Inhibitors of Intracellular RyR2 Calcium Release Channels as Therapeutic Agents in Arrhythmogenic Heart DiseasesTri Q Do, Björn C KnollmannPageof 33