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Seth L Alper

Showing results (131-140 of 257) with videos related to

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American Journal of Physiology. Cell Physiology|November 12, 2010
Native and recombinant Slc26a3 (downregulated in adenoma, Dra) do not exhibit properties of 2Cl-/1HCO3- exchangeSeth L Alper, Andrew K Stewart, David H Vandorpe, et al.
Blood Cells, Molecules & Diseases|November 12, 2021
Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell diseaseDavid H Vandorpe, Alicia Rivera, Boris E Shmukler, et al.
Science Advances|November 13, 2024
INF2 mutations cause kidney disease through a gain-of-function mechanismBalajikarthick Subramanian, Sarah Williams, Sophie Karp, et al.
Neuron|January 6, 2022
Brain ventricles as windows into brain development and diseasePhan Q Duy, Pasko Rakic, Seth L Alper, et al.
American Journal of Hematology|March 16, 2017
Erythrocytes from hereditary xerocytosis patients heterozygous for KCNN4 V282M exhibit increased spontaneous Gardos channel-like activity inhibited by senicapocAlicia Rivera, David H Vandorpe, Boris E Shmukler, et al.
Translational Research : the Journal of Laboratory and Clinical Medicine|April 14, 2023
Computational drug repositioning of clopidogrel as a novel therapeutic option for focal segmental glomerulosclerosisChristoph A Gebeshuber, Lisa Daniel-Fischer, Heinz Regele, et al.
Kidney International|August 24, 2024
Differing sensitivities to angiotensin converting enzyme inhibition of kidney disease mediated by APOL1 high-risk variants G1 and G2Esilida Sula Karreci, Sonako Jacas, Olivia Donovan, et al.
Biorxiv : the Preprint Server for Biology|June 25, 2024
Missense Mutant Gain-of-Function Causes Inverted Formin 2 (INF2)-Related Focal Segmental Glomerulosclerosis (FSGS)Balajikarthick Subramanian, Sarah Williams, Sophie Karp, et al.
Pflugers Archiv : European Journal of Physiology|February 16, 2022
Purinergic signaling is essential for full Psickle activation by hypoxia and by normoxic acid pH in mature human sickle red cells and in vitro-differentiated cultured human sickle reticulocytesDavid H Vandorpe, Alicia Rivera, Markus Ganter, et al.
American Journal of Physiology. Cell Physiology|May 20, 2011
SLC26 anion exchangers of guinea pig pancreatic duct: molecular cloning and functional characterizationAndrew K Stewart, Boris E Shmukler, David H Vandorpe, et al.
Pageof 26

Showing results (131-140 of 257) with videos related to

Sort By:
Pageof 26
American Journal of Physiology. Cell Physiology|November 12, 2010
Native and recombinant Slc26a3 (downregulated in adenoma, Dra) do not exhibit properties of 2Cl-/1HCO3- exchangeSeth L Alper, Andrew K Stewart, David H Vandorpe, et al.
Blood Cells, Molecules & Diseases|November 12, 2021
Trpv1 and Trpa1 are not essential for Psickle-like activity in red cells of the SAD mouse model of sickle cell diseaseDavid H Vandorpe, Alicia Rivera, Boris E Shmukler, et al.
Science Advances|November 13, 2024
INF2 mutations cause kidney disease through a gain-of-function mechanismBalajikarthick Subramanian, Sarah Williams, Sophie Karp, et al.
Neuron|January 6, 2022
Brain ventricles as windows into brain development and diseasePhan Q Duy, Pasko Rakic, Seth L Alper, et al.
American Journal of Hematology|March 16, 2017
Erythrocytes from hereditary xerocytosis patients heterozygous for KCNN4 V282M exhibit increased spontaneous Gardos channel-like activity inhibited by senicapocAlicia Rivera, David H Vandorpe, Boris E Shmukler, et al.
Translational Research : the Journal of Laboratory and Clinical Medicine|April 14, 2023
Computational drug repositioning of clopidogrel as a novel therapeutic option for focal segmental glomerulosclerosisChristoph A Gebeshuber, Lisa Daniel-Fischer, Heinz Regele, et al.
Kidney International|August 24, 2024
Differing sensitivities to angiotensin converting enzyme inhibition of kidney disease mediated by APOL1 high-risk variants G1 and G2Esilida Sula Karreci, Sonako Jacas, Olivia Donovan, et al.
Biorxiv : the Preprint Server for Biology|June 25, 2024
Missense Mutant Gain-of-Function Causes Inverted Formin 2 (INF2)-Related Focal Segmental Glomerulosclerosis (FSGS)Balajikarthick Subramanian, Sarah Williams, Sophie Karp, et al.
Pflugers Archiv : European Journal of Physiology|February 16, 2022
Purinergic signaling is essential for full Psickle activation by hypoxia and by normoxic acid pH in mature human sickle red cells and in vitro-differentiated cultured human sickle reticulocytesDavid H Vandorpe, Alicia Rivera, Markus Ganter, et al.
American Journal of Physiology. Cell Physiology|May 20, 2011
SLC26 anion exchangers of guinea pig pancreatic duct: molecular cloning and functional characterizationAndrew K Stewart, Boris E Shmukler, David H Vandorpe, et al.
Pageof 26